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MYD88 NP_002459.2:p.L265P

Known as: MYD88 L265P, MYD88 p.L265P, NP_002459.2:p.Leu265Pro 
A change in the amino acid residue at position 265 in the myeloid differentiation primary response protein MyD88 where leucine has been replaced by… Expand
National Institutes of Health

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Highly Cited
2013
Highly Cited
2013
By whole-genome and/or Sanger sequencing, we recently identified a somatic mutation (MYD88 L265P) that stimulates nuclear factor… Expand
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Highly Cited
2013
Highly Cited
2013
Myeloid differentiation factor 88 (MYD88) L265P somatic mutation is highly prevalent in Waldenström macroglobulinemia (WM) and… Expand
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Highly Cited
2013
Highly Cited
2013
We evaluated the MYD88 L265P mutation in Waldenström’s macroglobulinemia (WM) and B-cell lymphoproliferative disorders by… Expand
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Highly Cited
2012
Highly Cited
2012
BACKGROUND Waldenström's macroglobulinemia is an incurable, IgM-secreting lymphoplasmacytic lymphoma (LPL). The underlying… Expand
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Highly Cited
2011
Highly Cited
2011
Primary central nervous system lymphoma (PCNSL) is a special lymphoma entity. Although being a rare disease, the incidence of… Expand
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Highly Cited
2008
Highly Cited
2008
Toll-like receptor 4 (TLR4) induces two distinct signaling pathways controlled by the TIRAP-MyD88 and TRAM-TRIF pairs of adaptor… Expand
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Highly Cited
2008
Highly Cited
2008
Brucella abortus is a facultative intracellular bacterium that infects humans and domestic animals. The enhanced susceptibility… Expand
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Highly Cited
2007
Highly Cited
2007
The innate immune system relies on evolutionally conserved Toll-like receptors (TLRs) to recognize diverse microbial molecular… Expand
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Highly Cited
2001
Highly Cited
2001
The recognition of microbial pathogens by the innate immune system involves Toll-like receptors (TLRs), which recognize pathogen… Expand
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Highly Cited
1998
Highly Cited
1998
MyD88 has a modular organization, an N-terminal death domain (DD) related to the cytoplasmic signaling domains found in many… Expand
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