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IEM 1460

Known as: IEM-1460 
 
National Institutes of Health

Papers overview

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Highly Cited
2012
Highly Cited
2012
Glutamatergic synapses in early postnatal development transiently express calcium-permeable AMPA receptors (CP-AMPARs). Although… Expand
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Highly Cited
2011
Highly Cited
2011
Fast-spiking interneurons (FSIs) can exert powerful control over striatal output, and deficits in this cell population have been… Expand
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2011
2011
Peripheral inflammation alters AMPA receptor (AMPAR) subunit trafficking and increases AMPAR Ca(2+) permeability at synapses of… Expand
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Highly Cited
2010
Highly Cited
2010
It is well established that long-term potentiation (LTP), a paradigm for learning and memory, results in a stable enlargement of… Expand
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2010
2010
Overactivity of striatal alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) glutamate receptors is implicated in the… Expand
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2009
2009
Ca2+ influx via GluR2-lacking Ca2+-permeable AMPA glutamate receptors (CP-AMPARs) can trigger changes in synaptic efficacy in… Expand
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2005
2005
1. Antagonizing glutamatergic neurotransmission by blockade of AMPA-type glutamate receptors (GluR) is a promising… Expand
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1999
1999
Dicationic adamantane derivative, IEM-1460, which selectively blocks GluR2-lacking, Ca2+-permeable alpha-amino-3-hydroxy-5-methyl… Expand
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1999
1999
The properties of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA) receptors were examined in various cell types… Expand
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Highly Cited
1997
Highly Cited
1997
1. The effects of two adamantane derivatives, 1-trimethylammonio-5-(1-adamantane-methyl-ammoniopentane dibromide) (IEM-1460) and… Expand
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