IEM 1460

Known as: IEM-1460 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1959-2017
024619592017

Papers overview

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2013
2013
  • Rossiiskii fiziologicheskii zhurnal imeni I.M…
  • 2013
Intramuscular (i. m.) administration in the minimum effective dose (MED) of central analgesics of fentanyl and dipyrone… (More)
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2011
2011
Fast-spiking interneurons (FSIs) can exert powerful control over striatal output, and deficits in this cell population have been… (More)
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Highly Cited
2010
Highly Cited
2010
It is well established that long-term potentiation (LTP), a paradigm for learning and memory, results in a stable enlargement of… (More)
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2009
2009
Ca(2+) influx via GluR2-lacking Ca(2+)-permeable AMPA glutamate receptors (CP-AMPARs) can trigger changes in synaptic efficacy in… (More)
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2007
2007
Ca2+-permeable AMPA receptors (AMPARs) are expressed throughout the adult CNS but yet their role in development is poorly… (More)
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2005
2005
1. Antagonizing glutamatergic neurotransmission by blockade of AMPA-type glutamate receptors (GluR) is a promising… (More)
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1999
1999
Dicationic adamantane derivative, IEM-1460, which selectively blocks GluR2-lacking, Ca2+-permeable alpha-amino-3-hydroxy-5-methyl… (More)
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1999
1999
The properties of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA) receptors were examined in various cell types… (More)
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Highly Cited
1997
Highly Cited
1997
1. The effects of two adamantane derivatives, 1-trimethylammonio-5-(1-adamantane-methyl-ammoniopentane dibromide) (IEM-1460) and… (More)
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1996
1996
1. The mechanisms by which four adamantane derivatives (IEM-1857, -1592, -1460 and -1754) block the open NMDA-activated channel… (More)
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