IAANS

Known as: 2-(4'-(2''-iodoacetamido)phenyl)aminonaphthalene-6-sulfonic acid, 2-Naphthalenesulfonic acid, 6-((4-((iodoacetyl)amino)phenyl)amino)- 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1975-2016
01219752016

Papers overview

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2012
2012
The contractile response of the heart can be altered by disease-related protein modifications to numerous contractile proteins… (More)
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2009
2009
Recently four new hypertrophic cardiomyopathy mutations in cardiac troponin C (cTnC) (A8V, C84Y, E134D, and D145E) were reported… (More)
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1998
1998
The goal of this study is to characterize the kinetic mechanism of Ca2+ activation and inactivation of cardiac troponin C (cTnC… (More)
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1997
1997
Phosphorylation of two adjacent serine residues in the unique N-terminal extension of cardiac muscle troponin I (cTnI) is known… (More)
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1997
1997
During beta-adrenergic stimulation of the heart, there is a decrease in myofilament Ca2+ sensitivity mediated by the protein… (More)
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1997
1997
The goal of the current study was to generate recombinant cTnC proteins with single Cys residues as sites for attachment of… (More)
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1997
1997
The kinetics of the binding of Ca2+ to the single regulatory site of cardiac muscle troponin was investigated by using troponin… (More)
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1997
1997
A monocysteine mutant of cardiac muscle troponin I, cTnI(S5C/C81I/C98S), was generated from a mouse cTnI cDNA clone and expressed… (More)
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1997
1997
It is known that intracellular pH drops rapidly after the onset of ischemia in cardiac muscle and may play some role in the rapid… (More)
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1992
1992
The results of work by several investigators indicate that crossbridge attachment serves as a positive feedback mechanism that… (More)
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