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Hypoxic-Ischemic Encephalopathy

Known as: Ischemic Hypoxic Encephalopathy, Encephalopathies, Hypoxic-Ischemic, Anoxic encephalopathy 
Ischemic brain damage in which the entire brain is deprived of oxygen. It may be fatal or lead to long term disabilities including developmental… 
National Institutes of Health

Related topics

Related topics

8 relations
BrainCentral Nervous SystemEncephalopathiesHypoxic Brain Damage

Broader (1)

Hypoxia-Ischemia, Brain

Papers overview

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Highly Cited
2011
Highly Cited
2011
Human Umbilical Cord Blood Cells Restore Brain Damage Induced Changes in Rat Somatosensory Cortex
Intraperitoneal transplantation of human umbilical cord blood (hUCB) cells has been shown to reduce sensorimotor deficits after… 
Review
2006
Review
2006
"Indeterminate" microvolt T-wave alternans tests predict high risk of death or sustained ventricular arrhythmias in patients with left ventricular dysfunction.
Review
2003
Review
2003
Akt is a molecular target for signal transduction therapy in brain ischemic insult.
Growth factors including insulin-like growth factor-1 (IGF-1) promote cell survival in ischemic brain injury. Stimulation of IGF… 
Highly Cited
1999
Highly Cited
1999
Striatal outflow of adenosine, excitatory amino acids, gamma-aminobutyric acid, and taurine in awake freely moving rats after middle cerebral artery occlusion: correlations with neurological deficit…
BACKGROUND AND PURPOSE While a number of studies have investigated transmitter outflow in anesthetized animals after middle… 
Highly Cited
1996
Highly Cited
1996
The role of the cyclic AMP-responsive element binding protein (CREB) in hypoxic-ischemic brain damage and repair.
Highly Cited
1989
Highly Cited
1989
Role for gamma-aminobutyric acid in selective vulnerability in gerbils.
We tested the efficacy of various putative neuroprotective agents in the gerbil model of delayed neuronal death. The selective… 
Highly Cited
1988
Highly Cited
1988
Neurotoxicity of N-methyl-D-aspartate is markedly enhanced in developing rat central nervous system.
The neurotoxic lesion produced by direct injection of 25 nmol of N-methyl-D-aspartate (NMDA) into the corpus striatum of 7-day… 
Highly Cited
1987
Highly Cited
1987
Effect of clot removal at 24 hours on chronic vasospasm after SAH in the primate model.
The efficacy of complete clot removal 24 hours after subarachnoid hemorrhage (SAH) in the prevention of chronic cerebral… 
Highly Cited
1986
Highly Cited
1986
A reversible type of neuronal injury following ischemia in the gerbil hippocampus.
The Mongolian gerbil is known to develop delayed neuronal death in the hippocampus following brief forebrain ischemia (Brain Res… 
Highly Cited
1980
Highly Cited
1980
The relationship of ischemic contracture of vascular reperfusion in the isolated rat heart.
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