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Neuronal and Glial Apoptosis after Traumatic Spinal Cord Injury

The hypothesis that apoptosis dependent on active protein synthesis contributes to the neuronal and glial cell death, as well as to the neurological dysfunction, induced by mild-to-moderate severity traumatic insults to the rat spinal cord is supported.
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Transplanted embryonic stem cells survive, differentiate and promote recovery in injured rat spinal cord

Transplantation approaches using cellular bridges, fetal central nervous system cells, fibroblasts expressing neurotrophin-3 (ref. 6), hybridoma cells expressing inhibitory protein-blocking…
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Oligodendrocytes from forebrain are highly vulnerable to AMPA/kainate receptor-mediated excitotoxicity

The data suggest that oligodendrocytes share with neurons a high vulnerability to AMPA/kainate receptor-mediated death, a mechanism that may contribute to white matter injury in CNS disease.
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Physiological and pathophysiological roles of excitatory amino acids during central nervous system development

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Embryonic stem cells differentiate into oligodendrocytes and myelinate in culture and after spinal cord transplantation.

The ability of ES cell-derived oligodendrocytes to myelinate axons in culture and to replace lost myelin in the injured adult CNS is demonstrated.
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Spinal-cord injury

An overview of the newer therapeutic interventions employed in the care of the spinal cord injured individual and the theoretical rationale supporting them is presented.
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Chondroitin sulfate proteoglycans inhibit oligodendrocyte myelination through PTPσ

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Functional reorganization and stability of somatosensory-motor cortical topography in a tetraplegic subject with late recovery

The functional organization of somatosensory and motor cortex was investigated in an individual with a high cervical spinal cord injury, a 5-year absence of nearly all sensory/motor function at and…
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Robust CNS regeneration after complete spinal cord transection using aligned poly-L-lactic acid microfibers.

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Tissue-sparing effect of x-ray microplanar beams particularly in the CNS: is a bystander effect involved?

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