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Flufenamate

National Institutes of Health

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aluminum flufenamate

Papers overview

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2006
2006
Inhibition of Hypertonicity-Induced Cation Channels Sensitizes HeLa Cells to Shrinkage-Induced Apoptosis
Hypertonicity-induced cation channels (HICCs) are an effective mechanism of regulatory volume increase (RVI), which is a… 
1998
1998
The substrate recognition domain in the Na+/dicarboxylate and Na+/sulfate cotransporters is located in the carboxy-terminal portion of the protein.
1995
1995
Action of diphenylamine carboxylate derivatives, a family of non-steroidal anti-inflammatory drugs, on [Ca2+]i and Ca2+-activated channels in neurons
1995
1995
Adenosine 3′,5′-cyclic monophosphate and the self-priming effect of gonadotrophin-releasing hormone
1991
1991
Prostaglandin H synthase: perturbation of the tyrosyl radical as a probe of anticyclooxygenase agents.
EPR spectroscopy was used to study the effects of various nonsteroidal anti-inflammatory agents on the peroxidase-related tyrosyl… 
Highly Cited
1982
Highly Cited
1982
Evidence for a mediator role of thromboxane A2 in the myotropic action of leukotriene B4 (LTB4) on the guinea-pig lung.
Review
1982
Review
1982
Survey of antiinflammatory agents and related drugs as inhibitors of ovulation in the rabbit.
1977
1977
Elimination of inhibition in euglobulin fibrinolysis by use of flufenamate: involvement of C1-inactivator.
The fibrinolytic activity of euglobulin fractions prepared from human morning plasma and assayed on fibrin plates is strongly… 
1974
1974
Anti-complement effects of two anti-inflammatory agents. Niflumic and flufenamic acids.
Highly Cited
1968
Highly Cited
1968
Antagonism by Fenamates of Prostaglandin F2α and of Slow Reacting Substance on Human Bronchial Muscle
HISTAMINE, slow reacting substance in anaphylaxis (SRS-A) and prostaglandin F2α (PGF2α) are among substances obtained from lungs… 
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