FR 167653

Known as: FR167653 
 
National Institutes of Health

Papers overview

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Highly Cited
2009
Highly Cited
2009
BACKGROUND Recent studies have implicated the activation of stress-activated mitogen-activated protein kinase (MAPK) p38 in… (More)
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Highly Cited
2007
Highly Cited
2007
BACKGROUND Current evidence indicates that p38 mitogen-activated protein kinase activation in spinal microglia contributes to the… (More)
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2007
2007
The objective is to assess the effect of TNF-alpha inhibition on urinary albumin excretion in experimental diabetic rats. Male… (More)
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2007
2007
STUDY DESIGN Immunohistochemical and behavioral study using rat models of lumbar disc herniation and cauda equina syndrome… (More)
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2006
2006
OBJECTIVE p38 MAP kinase (p38 MAPK) and c-Jun NH2-terminal kinase (JNK) have been implicated in the pathophysiology of heart… (More)
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2005
2005
Alterations in the intracellular signal transduction pathway in primary afferents may contribute to pain hypersensitivity. We… (More)
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Highly Cited
2003
Highly Cited
2003
OBJECTIVE FR167653 is a potent inhibitor of p38 mitogen-activated protein kinase (MAPK) and inhibits tumor necrosis factor alpha… (More)
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2002
2002
BACKGROUND Several studies have implicated the mitogen-activated protein kinase (MAPK) signal pathway in non-hepatic organ… (More)
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2002
2002
BACKGROUND Various types of chemokines/cytokines play important roles in ischaemia/reperfusion injury in kidneys. However, the… (More)
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2000
2000
p38 mitogen-activated protein kinase (MAPK) is involved in intracellular signals that regulate a variety of cellular responses… (More)
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