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FR 167653

Known as: FR167653 
 
National Institutes of Health

Papers overview

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Highly Cited
2009
Highly Cited
2009
Background:Recent studies have implicated the activation of stress-activated mitogen-activated protein kinase (MAPK) p38 in… Expand
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Highly Cited
2007
Highly Cited
2007
The objective is to assess the effect of TNF-α inhibition on urinary albumin excretion in experimental diabetic rats. Male Wistar… Expand
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Highly Cited
2006
Highly Cited
2006
OBJECTIVE p38 MAP kinase (p38 MAPK) and c-Jun NH2-terminal kinase (JNK) have been implicated in the pathophysiology of heart… Expand
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Highly Cited
2005
Highly Cited
2005
Podocytes play an important role in maintaining normal glomerular function and structure, and podocyte injury leads to… Expand
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Highly Cited
2005
Highly Cited
2005
&NA; Alterations in the intracellular signal transduction pathway in primary afferents may contribute to pain hypersensitivity… Expand
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Highly Cited
2003
Highly Cited
2003
OBJECTIVE FR167653 is a potent inhibitor of p38 mitogen-activated protein kinase (MAPK) and inhibits tumor necrosis factor alpha… Expand
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Highly Cited
2001
Highly Cited
2001
FR167653 was discovered as a cytokine production inhibitor, but its target molecule has remained unclear. We examined the effect… Expand
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Highly Cited
2000
Highly Cited
2000
Abstract—p38 mitogen-activated protein kinase (MAPK) is involved in intracellular signals that regulate a variety of cellular… Expand
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Highly Cited
1999
Highly Cited
1999
Roles of enterobacteria, nitric oxide (NO) and neutrophil in indomethacin-induced small intestinal lesions were examined in rats… Expand
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Highly Cited
1996
Highly Cited
1996
FR167653 (1-[7-(4-fluorophenyl)-1,2,3,4-tetrahydro-8-(4-pyridyl)pyrazolo[5-1-c] [1,2,4]triazin-2-yl]-2-phenylethanedione sulfate… Expand
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