FBXO32 gene

Known as: MUSCLE ATROPHY F-BOX, MAFbx, FBXO32 
This gene plays a role in protein ubiquitination and degradation.
National Institutes of Health

Papers overview

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Highly Cited
2011
Highly Cited
2011
Skeletal muscle atrophy occurs under a variety of conditions and can result from alterations in both protein synthesis and… (More)
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Review
2010
Review
2010
Activation of the PI3 kinase pathway can induce skeletal muscle hypertrophy, defined as an increase in skeletal muscle mass. In… (More)
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Highly Cited
2010
Highly Cited
2010
Resistance to TGF-β is frequently observed in ovarian cancer, and disrupted TGF-β/SMAD4 signaling results in the aberrant… (More)
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Highly Cited
2007
Highly Cited
2007
Polycomb-repressive complex 2 (PRC2)-mediated histone methylation plays an important role in aberrant cancer gene silencing and… (More)
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Highly Cited
2007
Highly Cited
2007
The hypothesis of the present study was that exposure of differentiated muscle cells to agonists of the AMP-activated protein… (More)
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Highly Cited
2007
Highly Cited
2007
Resistance training using lengthening (eccentric) contractions induces greater increases in muscle size than shortening… (More)
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Highly Cited
2007
Highly Cited
2007
Smoking causes multiple organ dysfunction. The effect of smoking on skeletal muscle protein metabolism is unknown. We… (More)
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Highly Cited
2005
Highly Cited
2005
Atrogin1/MAFbx is an ubiquitin ligase that mediates muscle atrophy in a variety of catabolic states. We recently found that H2O2… (More)
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Review
2003
Review
2003
Skeletal muscle atrophy occurs in multiple clinical settings, including cancer, AIDS and sepsis, and is caused in part by an… (More)
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Highly Cited
2001
Highly Cited
2001
Skeletal muscle adapts to decreases in activity and load by undergoing atrophy. To identify candidate molecular mediators of… (More)
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