ESYT2 gene

Known as: EXTENDED SYNAPTOTAGMIN 2, FAMILY WITH SEQUENCE SIMILARITY 62, MEMBER B, FAM62B 
 
National Institutes of Health

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Genes

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2017
2017
The STI and UBA Domains of UBQLN1 Are Critical Determinants of Substrate Interaction and Proteostasis.
There are five Ubiquilin proteins (UBQLN1-4, UBQLN-L), which are evolutionarily conserved and structurally similar. UBQLN… (More)
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2017
2017
Genetic Variants Within Key Nodes of the Cascade of Antipsychotic Mechanisms: Effects on Antipsychotic Response and Schizophrenia Psychopathology in a Naturalistic Treatment Setting in Two…
INTRODUCTION Schizophrenia (SCZ) is one of the most disabling psychiatric disorders. Genetic factors play an important role in… (More)
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2016
2016
A large-scale analysis of alternative splicing reveals a key role of QKI in lung cancer.
Increasing interest has been devoted in recent years to the understanding of alternative splicing in cancer. In this study, we… (More)
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2015
2015
Extended Synaptotagmin Interaction with the Fibroblast Growth Factor Receptor Depends on Receptor Conformation, Not Catalytic Activity.
We previously demonstrated that ESyt2 interacts specifically with the activated FGF receptor and is required for a rapid phase of… (More)
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2014
2014
Loss of Extended Synaptotagmins ESyt2 and ESyt3 does not affect mouse development or viability, but in vitro cell migration and survival under stress are affected
The Extended Synaptotagmins (Esyts) are a family of multi-C2 domain membrane proteins with orthologs in organisms from yeast to… (More)
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2012
2012
Proteomic Study of the Mucin Granulae in an Intestinal Goblet Cell Model
Goblet cells specialize in producing and secreting mucus with its main component, mucins. An inducible goblet-like cell line was… (More)
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2009
2009
Candidate genes associated with malignant pheochromocytomas by genome-wide expression profiling.
OBJECTIVE To improve our understanding of the molecular mechanisms involved in malignant pheochromocytoma by examining… (More)
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2007
2007
Two independent chromosomal rearrangements, a very small (550 kb) duplication of the 7q subtelomeric region and an atypical 17q11.2 (NF1) microdeletion, in a girl with neurofibromatosis.
Most patients with neurofibromatosis (NF1) are endowed with heterozygous mutations in the NF1 gene. Approximately 5% show an… (More)
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