E2F1 protein, human

Known as: RBBP3 protein, human, E2F transcription factor 1 protein, human, Retinoblastoma-Binding Protein 3 
Transcription factor E2F1 (437 aa, ~47 kDa) is encoded by the human E2F1 gene. This protein is involved in the regulation of apoptosis, gene… (More)

Topic mentions per year

Topic mentions per year

1993-2017
05101519932017

Papers overview

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Highly Cited
2006
Highly Cited
2006
The E2F family of transcription factors regulates basic cellular processes. Here, we take an unbiased approach towards… (More)
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Highly Cited
2004
Highly Cited
2004
Overexpression or amplification of ACTR (also named AIB1, RAC3, p/CIP, TRAM-1, and SRC-3), a member of the p160 family of… (More)
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Highly Cited
2001
Highly Cited
2001
Previous work has established a role for p53 in triggering apoptosis in response to DNA damage; p53 also induces apoptosis in… (More)
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2001
2001
The transcription factor E2F1 is a key component of cell cycle that acts to transactivate genes required for S phase entry. Thus… (More)
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Highly Cited
2000
Highly Cited
2000
The six members of the E2F family of transcription factors play a key role in the control of cell cycle progression by regulating… (More)
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Highly Cited
1999
Highly Cited
1999
Functional inactivation of the pRB pathway is a very frequent event in human cancer, resulting in deregulated activity of the E2F… (More)
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Highly Cited
1999
Highly Cited
1999
The transcription factor E2F-1 is important in the control of cell proliferation. Its activity must be tightly regulated in a… (More)
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Highly Cited
1999
Highly Cited
1999
The E2F family of transcription factors plays an essential role in promoting cell cycle progression, and one member of the family… (More)
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Highly Cited
1997
Highly Cited
1997
The E2F transcription factors are essential for regulating the correct timing of activation of several genes whose products are… (More)
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Highly Cited
1994
Highly Cited
1994
E2F-1, a member of the E2F transcription factor family, contributes to the regulation of the G1-to-S phase transition in higher… (More)
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