DL-threo-beta-benzyloxyaspartate

Known as: DL-TBOA, TBOA 
 
National Institutes of Health

Papers overview

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Highly Cited
2009
Highly Cited
2009
In Alzheimer's disease (AD), the impairment of declarative memory coincides with the accumulation of extracellular amyloid-beta… (More)
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2008
2008
Long-term changes in the efficacy of glutamatergic synaptic transmission in the striatal complex are proposed to underlie motor… (More)
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Highly Cited
2004
Highly Cited
2004
Nontransportable blockers of the glutamate transporters are important tools for investigating mechanisms of synaptic transmission… (More)
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2004
2004
There is a body of evidence implying the involvement of the central glutamatergic system in morphine dependence. In this study… (More)
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Highly Cited
2004
Highly Cited
2004
Clearance of extracellular glutamate is essential for limiting the activity of metabotropic glutamate receptors (mGluRs) at… (More)
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2003
2003
Maintenance of low extracellular glutamate ([Glu](O)) preventing excitotoxic cell death requires fast removal of glutamate from… (More)
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2003
2003
Release of preloaded [3H]D-aspartate in response to depolarization induced by N-methyl-D-aspartate (NMDA) or the endogenous… (More)
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2000
2000
Elevated levels of the excitotoxic amino acids, glutamate and aspartate, have been implicated in the pathogenesis of neuronal… (More)
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Highly Cited
1998
Highly Cited
1998
DL-threo-beta-Benzyloxyaspartate (DL-TBOA), a novel derivative of DL-threo-beta-hydroxyaspartate, was synthesized and examined as… (More)
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