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CGP 56999A

Known as: CGP-56999A, CGP56999A 
National Institutes of Health

Papers overview

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2011
2011
RationaleSensory and sensorimotor gating deficits are observed in schizophrenia. GABAB receptor deficiency is also detected in… 
2008
2008
Paired‐pulse depression (PPD), a short‐term neural plasticity, was studied in hippocampal CA1 of urethane‐anesthetized rats in… 
2006
2006
Spike backpropagation has been proposed to enhance dendritic depolarization and synaptic plasticity. However, relatively little… 
2005
2005
Summary:  Purpose: To determine whether hippocampal afterdischarges (ADs) and excitability changes were induced by γ‐aminobutyric… 
2003
2003
  • X. RenI. Módy
  • Journal of Biological Chemistry
  • 2003
  • Corpus ID: 53646170
γ-Hydroxybutyrate (GHB) naturally occurs in the brain, but its exogenous administration induces profound effects on the central… 
2003
2003
  • X. RenI. Módy
  • The Journal of biological chemistry
  • 2003
  • Corpus ID: 23463875
gamma-Hydroxybutyrate (GHB) naturally occurs in the brain, but its exogenous administration induces profound effects on the… 
Highly Cited
2000
Highly Cited
2000
In this study we show that single, physiologically-active and non-convulsive doses of the three GABA(B) receptor antagonists CGP… 
1998
1998
Highly Cited
1996
Highly Cited
1996
1 The effects of various GABA receptor ligands on the electrically‐evoked release of endogenous GABA, glutamate and substance P…