CGP 56999A

Known as: CGP-56999A, CGP56999A 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1996-2012
01219962012

Papers overview

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2011
2011
Sensory and sensorimotor gating deficits are observed in schizophrenia. GABAB receptor deficiency is also detected in the… (More)
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2010
2010
Whether seizures in the developing brain cause long-term changes in the mature brain has been debated. We tested the hypothesis… (More)
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2008
2008
Paired-pulse depression (PPD), a short-term neural plasticity, was studied in hippocampal CA1 of urethane-anesthetized rats in… (More)
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2006
2006
Spike backpropagation has been proposed to enhance dendritic depolarization and synaptic plasticity. However, relatively little… (More)
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2006
2006
Rats were injected (i.p.) once daily with either 1 mg/kg CGP 56999A, a gamma-aminobutyric acid(B) (GABA(B)) receptor antagonist… (More)
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2003
2003
gamma-Hydroxybutyrate (GHB) naturally occurs in the brain, but its exogenous administration induces profound effects on the… (More)
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2000
2000
In this study we show that single, physiologically-active and non-convulsive doses of the three GABA(B) receptor antagonists CGP… (More)
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1997
1997
CGP 56999A ([3-[1-(R)-[(3-cyclohexylmethyl)hydroxyphosphinyl]-2-(S)- hydroxy-propyl] amino]ethyl]-benzoic acid) is a potent GABAB… (More)
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1996
1996
1. The effects of various GABA receptor ligands on the electrically-evoked release of endogenous GABA, glutamate and substance P… (More)
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