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CCL26 protein, human

Known as: eotaxin 3, human, Small Inducible Cytokine A26, C-C Motif Chemokine 26 
C-C motif chemokine 26 (94 aa, ~11 kDa) is encoded by the human CCL26 gene. This protein plays a role in eosinophil and basophil chemotaxis.
National Institutes of Health

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16 relations
Biologic DevelopmentCCL26 geneCCL26 wt AlleleCentral Nervous System

Broader (1)

beta-Chemokines

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2017
2017
Gene Expression Patterns in Distinct Endoscopic Findings for Eosinophilic Gastritis in Children.
2014
2014
Demethylation of the Human Eotaxin-3 Gene Promoter Leads to the Elevated Expression of Eotaxin-3
DNA demethylation has been primarily studied in the context of development biology, cell fate, and cancer, with less attention on… 
2012
2012
STAT6 phosphorylation inhibitors block eotaxin-3 secretion in bronchial epithelial cells.
2012
2012
Leukotriene D4 and Interleukin-13 Cooperate to Increase the Release of Eotaxin-3 by Airway Epithelial Cells
Introduction Airway epithelial cells play a central role in the physiopathology of asthma. They release eotaxins when treated… 
2009
2009
Chitin Stimulates Expression of Acidic Mammalian Chitinase and Eotaxin-3 by Human Sinonasal Epithelial Cells in Vitro
Background Sinonasal epithelial cells participate in host defense by initiating innate immune mechanisms against potential… 
2008
2008
Ontogeny of the eotaxins in human lung.
The ontogeny of the C-C chemokines eotaxin-1, eotaxin-2, and eotaxin-3 has not been fully elucidated in human lung. We explored a… 
Highly Cited
2005
Highly Cited
2005
Eotaxin-3/CCL26 gene expression in intestinal epithelial cells is up-regulated by interleukin-4 and interleukin-13 via the signal transducer and activator of transcription 6.
2005
2005
Comparison of plasma eotaxin family level in aspirin-induced and aspirin-tolerant asthma patients.
STUDY OBJECTIVE Eosinophilic infiltration of airway tissue is a central feature of aspirin-induced asthma (AIA). Eotaxins belong… 
2005
2005
dsRNA enhances eotaxin-3 production through interleukin-4 receptor upregulation in airway epithelial cells
The exacerbation of asthma during viral infections is mainly explained by neutrophils infiltrating into the airways. However… 
2004
2004
Upregulation of interleukin-4 receptor by interferon-gamma: enhanced interleukin-4-induced eotaxin-3 production in airway epithelium.
Airway epithelial cells produce a number of chemokines, including eotaxins. Among the three known eotaxins, T helper (Th) type 2… 
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