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C1QA gene

Known as: COMPLEMENT COMPONENT C1q, A CHAIN, SERUM C1q, Complement Component 1, q Subcomponent, A Chain Gene 
This gene plays a role in the classical complement pathway.
National Institutes of Health

Related topics

Related topics

3 relations
C1QBP geneComplement ActivationComplement C1q Subcomponent Subunit A

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2019
Highly Cited
2019
Complement 3+-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia
Astrogliosis and activation of microglia are hallmarks of prion diseases in humans and animals. Both were viewed to be rather… 
Highly Cited
2015
Highly Cited
2015
APOE Stabilization by Exercise Prevents Aging Neurovascular Dysfunction and Complement Induction
Aging is the major risk factor for neurodegenerative diseases such as Alzheimer's disease, but little is known about the… 
2012
2012
Homozygous A polymorphism of the complement C1qA276 correlates with prolonged overall survival in patients with diffuse large B cell lymphoma treated with R-CHOP
BackgroundThe precise mechanism of action for rituximab (R) is not fully elucidated. Besides antibody-dependent cellular… 
2012
2012
Transformative binding and release of gold guests from a self-assembled Cu8L4 tube.
The highly specific binding, transformation and protection of chemical compounds are functions associated with biomolecular… 
Highly Cited
2009
Highly Cited
2009
Differential gene expression in LPS/IFNγ activated microglia and macrophages: in vitro versus in vivo
Two different macrophage populations contribute to CNS neuroinflammation: CNS‐resident microglia and CNS‐infiltrating peripheral… 
2009
2009
Influenza A virus M1 blocks the classical complement pathway through interacting with C1qA.
The matrix (M1) protein of influenza A virus is a conserved multifunctional protein that plays essential roles in regulating the… 
Highly Cited
2007
Highly Cited
2007
Bandwidth and Fidelity on the NEO-Five Factor Inventory: Replicability and Reliability of Saucier's (1998) Item Cluster Subcomponents
Many users of the NEO-Five Factor Inventory (NEO-FFI; Costa & McCrae, 1992) are unaware that Saucier (1998) developed item… 
2000
2000
Molecular, genetic and epidemiologic studies on selective complete C1q deficiency in Turkey.
Highly Cited
1999
Highly Cited
1999
C1q binding to liposomes is surface charge dependent and is inhibited by peptides consisting of residues 14-26 of the human C1qA chain in a sequence independent manner.
Highly Cited
1976
Highly Cited
1976
C1 subcomponent conplexes in normal and pathological sera studied by crossed immunoelectrophoresis.
Selected pathological sera gave three molecular species of C1s protein on crossed immunoelectrophoresis in the presence of… 
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