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BZA 5B

Known as: BZA-5B 
 
National Institutes of Health

Papers overview

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Highly Cited
2002
Highly Cited
2002
Hepatitis delta virus (HDV) causes both acute and chronic liver disease throughout the world. Effective medical therapy is… Expand
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Highly Cited
1997
Highly Cited
1997
Mutations in Ras family members that confer oncogenic potential are frequently observed in specific human cancers. We report that… Expand
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1997
1997
The mevalonate (MVA) pathway is involved in cell proliferation. We investigated drugs acting at different enzymatic steps on rat… Expand
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1997
1997
Ras proteins have been implicated in transducing cellular responses to DNA damaging agents. We used BZA-5B, an inhibitor of Ras… Expand
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Highly Cited
1996
Highly Cited
1996
Adult rat ventricular myocytes and cardiac microvascular endothelial cells (CMEC) both express an inducible nitric oxide synthase… Expand
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1996
1996
Benzodiazepine (BZA)-5B, a CAAX farnesyl-transferase inhibitor, was previously shown to block the farnesylation of H-Ras and to… Expand
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1996
1996
Interferons (IFN) and lipopolysaccharide (LPS) cause multiple changes in isoprenoid-modified proteins in murine macrophages, the… Expand
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1995
1995
BZA-5B is a peptidomimetic inhibitor of protein farnesylation in mammalian cells. We have examined the specificity of this… Expand
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Highly Cited
1995
Highly Cited
1995
BZA-5B, a benzodiazepine peptidomimetic, inhibits CAAX farnesyltransferase (FTase) and blocks attachment of farnesyl groups to… Expand
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1994
1994
A benzodiazepine peptidomimetic, BZA-5B, inhibits farnesylation of H-Ras and normalizes the morphology of Rat-1 cells transformed… Expand
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