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BZA 5B

Known as: BZA-5B 
 
National Institutes of Health

Papers overview

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Highly Cited
2002
Highly Cited
2002
ABSTRACT Hepatitis delta virus (HDV) causes both acute and chronic liver disease throughout the world. Effective medical therapy… Expand
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Highly Cited
1997
Highly Cited
1997
Mutations in Ras family members that confer oncogenic potential are frequently observed in specific human cancers. We report that… Expand
Highly Cited
1997
Highly Cited
1997
The mevalonate (MVA) pathway is involved in cell proliferation. We investigated drugs acting at different enzymatic steps on rat… Expand
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1997
1997
Ras proteins have been implicated in transducing cellular responses to DNA damaging agents. We used BZA-5B, an inhibitor of Ras… Expand
Highly Cited
1996
Highly Cited
1996
Adult rat ventricular myocytes and cardiac microvascular endothelial cells (CMEC) both express an inducible nitric oxide synthase… Expand
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Highly Cited
1996
Highly Cited
1996
Interferons (IFN) and lipopolysaccharide (LPS) cause multiple changes in isoprenoid‐modified proteins in murine macrophages, the… Expand
Highly Cited
1996
Highly Cited
1996
Benzodiazepine (BZA)-5B, a CAAX farnesyl-transferase inhibitor, was previously shown to block the farnesylation of H-Ras and to… Expand
Highly Cited
1995
Highly Cited
1995
BZA-5B, a benzodiazepine peptidomimetic, inhibits CAAX farnesyltransferase (FTase) and blocks attachment of farnesyl groups to… Expand
Highly Cited
1995
Highly Cited
1995
BZA-5B is a peptidomimetic inhibitor of protein farnesylation in mammalian cells. We have examined the specificity of this… Expand
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Highly Cited
1994
Highly Cited
1994
A benzodiazepine peptidomimetic, BZA-5B, inhibits farnesylation of H-Ras and normalizes the morphology of Rat-1 cells transformed… Expand