Acute Kidney Tubular Necrosis

Known as: Kidney Tubular Necrosis, Acute, acute renal tubular necrosis, Kidney Tubular Necrosis, Acute [Disease/Finding] 
Acute kidney failure resulting from destruction of EPITHELIAL CELLS of the KIDNEY TUBULES. It is commonly attributed to exposure to toxic agents or… (More)
National Institutes of Health

Papers overview

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Highly Cited
2009
Highly Cited
2009
When patients develop acute kidney injury, a small fraction of them will develop end-stage renal disease later. The severity of… (More)
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Highly Cited
2008
Highly Cited
2008
Understanding the mechanisms of nephron repair is critical for the design of new therapeutic approaches to treat kidney disease… (More)
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Review
2008
Review
2008
UNLABELLED Acute renal failure (ARF), recently renamed acute kidney injury (AKI), is a relatively frequent problem, occurring in… (More)
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Highly Cited
2007
Highly Cited
2007
Ischemia/reperfusion injury (IRI) may activate innate immunity through the engagement of TLRs by endogenous ligands. TLR4… (More)
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Highly Cited
2004
Highly Cited
2004
BACKGROUND Interleukin-18 (IL-18) is a mediator of ischemic acute tubular necrosis (ATN) in mice. METHODS IL-18 was measured in… (More)
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Highly Cited
2003
Highly Cited
2003
BACKGROUND Renal failure and toxic acute tubular necrosis (ATN) may be seen following exposure to a variety of therapeutic agents… (More)
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Highly Cited
2002
Highly Cited
2002
BACKGROUND Traditional blood and urine markers for the diagnosis of various renal diseases are insensitive and nonspecific… (More)
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Highly Cited
2002
Highly Cited
2002
Having recently described the injurious role of caspase-1-mediated production of the proinflammatory cytokine IL-18 in ischemic… (More)
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Highly Cited
1996
Highly Cited
1996
There are very limited data on overall epidemiology of ARF. It is crucial to know the incidence, etiology and clinical feature of… (More)
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Highly Cited
1977
Highly Cited
1977
Irreversible ischemic myocardial cell injury developes in an increasing number of cells as the duration of coronary occlusion is… (More)
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