Acecainide

Known as: Benzamide, 4-(acetylamino)-N-(2-(diethylamino)ethyl)-, N Acetylprocainamide, N-acetylprocainamide 
A major metabolite of PROCAINAMIDE. Its anti-arrhythmic action may cause cardiac toxicity in kidney failure.
National Institutes of Health

Papers overview

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2002
2002
A cDNA coding a novel organic cation transporter, hOCT2-A, was isolated from human kidney. The hOCT2-A cDNA is an alternatively… (More)
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1997
1997
OBJECTIVE T cells treated with DNA methylation inhibitors overexpress lymphocyte function-associated antigen 1 (LFA-1), which… (More)
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1997
1997
We studied the genotypes of polymorphic N-acetyltransferase (NAT2) in 145 Japanese subjects by the polymerase chain reaction… (More)
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Review
1993
Review
1993
Common clinical features in drug-induced torsade de pointes include hypokalemia and cycle-length prolongation just prior to… (More)
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1983
1983
To investigate the absorption of aerosolized drugs from the rat lung, anesthetized animals were allowed to inhale through a… (More)
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1983
1983
The hypothesis that basic drugs can compete for active tubular secretion by the kidney was tested in six healthy volunteers by… (More)
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1983
1983
The acetylation pathway of drug metabolism is the pathway by which aromatic amines and hydrazines are metabolized. Hydralazine… (More)
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1975
1975
N-Acetylprocainamide (NAPA) and procainamide plasma and urine concentrations were determined by thin-layer chromatography (TLC… (More)
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1975
1975
Absorption of a single oral dose of N-acetylprocainamide (NAPA) was studied in 3 normal subjects. Approximately 85% of the oral… (More)
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1975
1975
Pharmacokinetics of procainamide were studied in healthy volunteers after single doses intravenously and orally as conventional… (More)
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