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ACTH (5-24)

Known as: 5-24 ACTH, ACTH(5-24), corticotropin-(5-24)-peptide 
National Institutes of Health

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2 relations

Broader (2)

CosyntropinPeptide Fragments

Papers overview

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1994
1994
Catalytic subunit of protein kinase A induces amylase release from streptolysin O-permeabilized parotid acini.
Amylase release from parotid acinar cells is a typical model of cAMP-mediated exocytosis. To obtain unequivocal data concerning… 
1992
1992
Protein kinase A modulates Ca(2+)- and protein kinase C-dependent amylase release in permeabilized rat pancreatic acini.
The role of protein kinase A (PKA) in the release of amylase from permeabilized pancreatic acini was investigated. Addition of… 
1988
1988
Evidence against direct involvement of cyclic AMP-dependent protein phosphorylation in the exocytosis of amylase.
To examine whether or not the activation of cyclic AMP-dependent protein kinase is coupled to the exocytosis of amylase from rat… 
1988
1988
Neuropeptide K-(1–24)-peptide: Storage and release by carcinoid tumors
1984
1984
A dual pathway for ACTH steroidogenic action in purified adrenocortical cells.
Isolated adrenal fasciculata cells were purified by centrifugation through a 0-50% hyperbolic gradient of PercollR. The dose… 
1980
1980
Effects of ACTH (corticotropin) analogues on steroidogenesis and cyclic AMP in rat adrenocortical cells. Evidence for two different steroidogenically responsive receptors.
Comparative studies on the mechanism of action of ACTH1-39 and ACTH5-24 [corticotropin-(1-39)- and corticotropin-(5-24)-peptides… 
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