ABT-089

Known as: A-87089.0, ABT 089, ABYT 089 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1997-2014
024619972014

Papers overview

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2015
2015
Nicotine withdrawal produces cognitive deficits that can predict relapse. Amelioration of these cognitive deficits emerges as a… (More)
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2014
2014
Levodopa-induced dyskinesias (LIDs) are a serious complication of levodopa therapy for Parkinson's disease for which there is… (More)
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2011
2011
OBJECTIVE To assess the safety and efficacy of ABT-089, a novel α(4)β(2) neuronal nicotinic receptor partial agonist, vs. placebo… (More)
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2011
2011
α4β2 Neuronal nicotinic receptors (NNRs) are implicated in the pathophysiology of attention-deficit/hyperactivity disorder (ADHD… (More)
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2009
2009
Numerous pharmaceutical efforts have targeted neuronal nicotinic receptors (nAChRs) for amelioration of cognitive deficits. While… (More)
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2006
2006
BACKGROUND This pilot study was designed to evaluate ABT-089, a neuronal nicotinic receptor partial agonist, as treatment for… (More)
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1998
1998
 Increased distractibility is associated with both Alzheimer’s disease and attention deficit disorder. The present study examined… (More)
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1997
1997
Accumulating preclinical and clinical evidence data suggests that compounds that selectively activate neuronal nicotinic… (More)
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1997
1997
ABT-089 [2-methyl-3-(2-(S)-pyrrolidinylmethoxy)pyridine dihydrochloride], a novel ligand at neuronal nicotinic acetylcholine… (More)
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1997
1997
2-Methyl-3-(2(S)-pyrrolidinylmethoxy)pyridine, ABT-089 (S-4), a member of the 3-pyridyl ether class of nicotinic acetylcholine… (More)
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