Ultrasonographic backscatter of the carotid artery wall in patients with HIV infection: A pilot study

@article{Giannattasio2010UltrasonographicBO,
  title={Ultrasonographic backscatter of the carotid artery wall in patients with HIV infection: A pilot study},
  author={Cristina Giannattasio and Monica Failla and Nicola Squillace and Alberto Dolara and Francesca Cesana and Francesca Sabbatini and Alessandra Bandera and Rita Facchetti and Dario Dozio and Andrea Gori and Giuseppe Mancia},
  journal={Blood Pressure},
  year={2010},
  volume={19},
  pages={344 - 350},
  url={https://api.semanticscholar.org/CorpusID:207471886}
}
Classic risk factors greatly affect cIMT than time of HIV infection, duration of antiretroviral therapy exposure and use of protease inhibitors, and IBS is a promising technique for the evaluation of arterial wall composition in HIV patients.
3 Citations

Brachial and central blood pressure in HIV-infected subjects

The study shows that the unfavorable CV risk profile associated with HIV infection includes an increase in both central BP and Aix, which seems to be favored by renal damage, which apparently has a role in the early stages of the disease.

Metabolic syndrome in human immunodeficiency virus-positive subjects: prevalence, phenotype, and related alterations in arterial structure and function.

HIV infection itself and highly active antiretroviral treatment, more than HIV infection per se, appeared to be responsible for the increased prevalence of metabolic syndrome and arterial function derangement.

Cardiovascular risks in HIV patients

Although the carotid intima-media thickness in HIV patients increased over 2 years of observation, there was no correlation to HIV-RNA or CD4, and the result is that treated HIV patients will carry an increased risk of future cardiovascular disease (CVD) (6,7).

Progression of Atherosclerosis as Assessed by Carotid Intima-Media Thickness in Patients With HIV Infection

Carotid IMT is higher in HIV patients than in age-matched control subjects and progresses much more rapidly than previously reported rates in non-HIV cohorts, suggesting that immunodeficiency and traditional coronary risk factors may contribute to atherosclerosis.

Brief report: carotid intima-media thickness in heavily pretreated HIV-infected patients.

It is suggested that lipid disturbances, mainly hypoHDLemia, may be involved in the early atherosclerotic process in HIV-infected patients.

Progression of carotid artery intima–media thickening in HIV-infected and uninfected adults

HIV infection and PI use did not contribute substantially to the rate of carotid IMT progression in the authors' matched study.

Colour-Doppler ultrasonography of carotid vessels in patients treated with antiretroviral therapy: a comparative study

These data confirm the higher prevalence of premature carotid lesions in the PI-treated patients, and a periodic ultrasonographic study of the vascular wall should be included in the follow-up of HIV infected patients.

Ultrasound B-mode imaging in observational studies of atherosclerotic progression.

Age, serum low density lipoprotein cholesterol, smoking, platelet aggregability, serum copper, serum selenium, and blood hemoglobin were the strongest predictors of 2-year increase of common carotid IMT.

An ultrasound-based comparative study on carotid plaques in HIV-positive patients vs. atherosclerotic and arteritis patients: atherosclerotic or inflammatory lesions?

The study evidenced that the ultrasonographic structure of the epi-aortic lesions in HIV-positive patients substantially differ from those of the plaques in atherosclerotic patients, although they share similar characteristics with patients affected by arteritis.
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