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response to paracetamol

Known as: response to acetaminophen 
Any process that results in a change in state or activity of a cell or an organism (in terms of movement, secretion, enzyme production, gene… 
National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2017
2017
Development of Gold Modified Disposable Pencil Graphite Electrode for the Electrochemical Investigation of Acetaminophen Present in Pharmaceutical formulations and Biological samples
A novel electrochemical sensor based on the electrodeposition of gold nanoparticles on disposable pencil graphite electrode… 
2016
2016
Reducing Hepatocyte Injury and Necrosis in Response to Paracetamol Using Noncoding RNAs
The liver performs multiple functions within the human body. It is composed of numerous cell types, which play important roles in… 
2015
2015
Adaptation to acetaminophen exposure elicits major changes in expression and distribution of the hepatic proteome
Acetaminophen overdose is the leading cause of acute liver failure. One dose of 10–15 g causes severe liver damage in humans… 
2015
2015
M1 muscarinic receptors modify oxidative stress response to acetaminophen-induced acute liver injury.
2015
2015
Circulating microRNAs Reveal Time Course of Organ Injury in a Porcine Model of Acetaminophen-Induced Acute Liver Failure
Acute liver failure is a rare but catastrophic condition which can progress rapidly to multi-organ failure. Studies investigating… 
2015
2015
Investigation of acetaminophen toxicity in HepG2/C3a microscale cultures using a system biology model of glutathione depletion
We have integrated in vitro and in silico information to investigate acetaminophen (APAP) and its metabolite N-acetyl-p… 
2013
2013
460: FEVER RESPONSE TO ACETAMINOPHEN IN CHILDREN WITH SEVERE TRAUMATIC BRAIN INJURY IS DOSE DEPENDENT
9.9 ± 2.2 mg/dL, p=0.41). Consistent with existing literature, patients with MOF and/or septic shock had higher serum… 
2013
2013
551 A PROTEOMIC ROAD MAP OF THE HepaRG CELL LINE: METABOLIC PATHWAYS PRESENT AND THEIR RESPONSE TO ACETAMINOPHEN TREATMENT
1988
1988
Deficiency in bilirubin UDP-glucuronyl transferase as a genetic determinant of acetaminophen toxicity.
Over 60% of the analgesic/antipyretic drug acetaminophen is eliminated by glucuronidation, which competes with a toxifying… 
Highly Cited
1985
Highly Cited
1985
Acetaminophen hepatotoxicity in vivo is not accompanied by oxidant stress.
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