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endobain E

National Institutes of Health

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2 relations

Broader (1)

Ouabain
analogs & derivatives

Papers overview

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2007
2007
The Expression of NMDA Receptor Subunits in Cerebral Cortex and Hippocampus is Differentially Increased by Administration of Endobain E, a Na+, K+-ATPase Inhibitor
Previous studies showed that endobain E, an endogenous Na+, K+-ATPase inhibitor, decreases dizocilpine binding to NMDA receptor… 
2005
2005
Modulation of Aspartate Release by Ascorbic Acid and Endobain E, an Endogenous Na+, K+-ATPase Inhibitor
The isolation of a soluble brain fraction which behaves as an endogenous ouabain-like substance, termed endobain E, has been… 
2004
2004
Allosteric modulation of [3H]dizocilpine binding to N-methyl-d-aspartate receptor by an endogenous Na+, K+-ATPase inhibitor: dependence on receptor activation
2004
2004
The Effect of Endogenous Modulator Endobain E on NMDA Receptor Is Interfered by Zn22+ but Is Independent of Modulation by Spermidine
A brain endogenous factor, termed endobain E, allosterically decreases [3H]dizocilpine binding to NMDA receptor. Such effect… 
2003
2003
A Comparative Study Between a Brain Na+,K+-ATPase Inhibitor (Endobain E) and Ascorbic Acid
In the search of Na+,K+-ATPase modulators, we have reported the isolation by gel filtration and HPLC of a brain fraction, termed… 
2002
2002
Metabotropic glutamate receptor involvement in phosphoinositide hydrolysis stimulation by an endogenous Na(+), K(+)-ATPase inhibitor and ouabain in neonatal rat brain.
2001
2001
[3H]Dizocilpine binding to N-methyl-d-aspartate (NMDA) receptor is modulated by an endogenous Na+, K+-ATPase inhibitor. Comparison with ouabain
2001
2001
An Endogenous Na+, K+-ATPase Inhibitor Enhances Phosphoinositide Hydrolysis in Neonatal but Not in Adult Rat Brain Cortex
The effect of an endogenous Na+, K+-ATPase inhibitor, termed endobain E, on phosphoinositide hydrolysis was studied in rat brain… 
1999
1999
A brain Na+, K+-ATPase inhibitor (endobain E) enhances norepinephrine release in rat hypothalamus
1998
1998
Na+,K+-ATPase interaction with a brain endogenous inhibitor (endobain E)
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