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UCF 101

Known as: UCF-101, UCF101 cpd 
National Institutes of Health

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2 relations

Broader (2)

PyrimidinonesThiones

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2014
2014
Upregulation of mitochondrial protease HtrA2/Omi contributes to manganese-induced neuronal apoptosis in rat brain striatum
2009
2009
UCF-101 mitigates streptozotocin-induced cardiomyocyte dysfunction: role of AMPK.
Diabetic heart disease contributes to the high mortality in diabetics, although effective clinical management is lacking. The… 
2009
2009
The protease inhibitor UCF‐101 ameliorates streptozotocin‐induced mouse cardiomyocyte contractile dysfunction in vitro: role of AMP‐activated protein kinase
5‐[5‐(2‐Nitrophenyl) furfuryliodine]‐1,3‐diphenyl‐2‐thiobarbituric acid (UCF‐101) is a protease inhibitor which was reported to… 
Highly Cited
2008
Highly Cited
2008
Participation of Omi Htra2 Serine-Protease Activity in the Apoptosis Induced by Cisplatin on SW480 Colon Cancer Cells
Abstract Apoptosis is triggered by two interconnected pathways, extrinsic and intrinsic. The intrinsic pathway is activated by… 
Highly Cited
2008
Highly Cited
2008
HtrA2/Omi Terminates Cytomegalovirus Infection and Is Controlled by the Viral Mitochondrial Inhibitor of Apoptosis (vMIA)
Viruses encode suppressors of cell death to block intrinsic and extrinsic host-initiated death pathways that reduce viral yield… 
2006
2006
High performance room temperature nematic liquid crystals based on laterally fluorinated isothiocyanato-tolanes
High birefringence and low viscosity isothiocyanates liquid crystal single compounds and eutectic mixtures, based solely on… 
2006
2006
The protease inhibitor Ucf-101 induces cellular responses independently of its known target, HtrA2/Omi
The protease inhibitor Ucf-101 induces cellular responses independently of its known target, HtrA2/Omi 
Highly Cited
2005
Highly Cited
2005
Role of Omi/HtrA2 in Apoptotic Cell Death After Myocardial Ischemia and Reperfusion
Background—Omi/HtrA2 is a proapoptotic mitochondrial serine protease involved in caspase-dependent as well as caspase-independent… 
Highly Cited
2004
Highly Cited
2004
Intramitochondrial serine protease activity of Omi/HtrA2 is required for caspase-independent cell death of human neutrophils
Intramitochondrial serine protease activity of Omi/HtrA2 is required for caspase-independent cell death of human neutrophils 
Highly Cited
2004
Highly Cited
2004
Omi/HtrA2 Promotes Cell Death by Binding and Degrading the Anti-apoptotic Protein ped/pea-15*
ped/pea-15 is a ubiquitously expressed 15-kDa protein featuring a broad anti-apoptotic function. In a yeast two-hybrid screen… 
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