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T-817MA

National Institutes of Health

Papers overview

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2014
2014
Effects of the neurotrophic agent T-817MA on oligomeric amyloid-β–induced deficits in long-term potentiation in the hippocampal CA1 subfield
2012
2012
T-817MA, a novel neurotrophic agent, ameliorates loss of GABAergic parvalbumin-positive neurons and sensorimotor gating deficits in rats transiently exposed to MK-801 in the neonatal period.
2011
2011
T-817MA, a neuroprotective agent, attenuates the motor and cognitive impairments associated with neuronal degeneration in P301L tau transgenic mice.
2011
2011
Protective effect of neurotrophic agent T-817MA against inner ear barotrauma in the guinea pig.
T-817MA (1-{3-[2-(1-benzothiophen-5-yl)ethoxy]propyl}azetidin-3-ol maleate), a neurotrophic compound newly synthesized for the… 
Review
2010
Review
2010
[Pharmacological properties of T-817MA, a novel neurotrophic agent, for treatment of Alzheimer's disease.].
  • T. Fukushima
  • Nihon yakurigaku zasshi. Folia pharmacologica…
  • 2010
  • Corpus ID: 22014487
要約：アルツハイマー病（AD）は進行性の神経変性疾患であり，アミロイドβ（Aβ）やタウの凝集・蓄積に起因する神経変性が病態に関与すると考えられている．T-817MAは，神経栄養因子様作用を有する低分子化合物であり，AD… 
2008
2008
T-817MA, a novel neurotrophic compound, ameliorates phencyclidine-induced disruption of sensorimotor gating
RationaleNeurodegenerative changes have been suggested to provide a basis for the pathophysiology of schizophrenia. T-817MA (1-{3… 
2008
2008
T-817MA, a novel neurotrophic compound, ameliorates phencyclidine-induced disruption of sensorimotor gating in rats
2008
2008
T-817MA facilitates adult neurogenesis and spatial memory in amyloid-β peptide infused rats (生体機能と創薬シンポジウム2008--生命システムにおける情報ネットワークの重要性を解く 講演要旨集)
2006
2006
T-817MA, a novel neurotrophic agent, improves sodium nitroprusside-induced mitochondrial dysfunction in cortical neurons
2005
2005
Attenuates Amyloid-β-induced Neurotoxicity and Promotes Neurite Outgrowth in Rat Cultured Central Nervous System Neurons
Section: Neuropharmacology This article has not been copyedited and formatted. The final version may differ from this version… 
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