SIN 1C

Known as: SIN-1C 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

1985-2015
012319852015

Papers overview

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1999
1999
SIN-1 has been used, in vitro, to simultaneously generate nitric oxide (*NO) and superoxide (O*-2). However, the pharmacological… (More)
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1998
1998
Previous studies have shown that SIN-1C (N-morpholinoiminoacetonitrile) can protect ischemic-reperfused myocardium. The aim of… (More)
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1996
1996
PURPOSE To evaluate the effect of intraocular administration of nitric oxide (NO) donors in the rabbit eye on intraocular… (More)
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Highly Cited
1995
Highly Cited
1995
Cardiac Ca2+ current (ICa) was shown to be regulated by cGMP in a number of different species. Recently, we found that the NO… (More)
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1995
1995
Reperfusion of ischemic myocardium may aggravate the ischemic state of injury and thus augment infarct size (reperfusion injury… (More)
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1994
1994
It was investigated whether morpholinosydnonimine (SIN-1), which spontaneously decomposes into NO and 3… (More)
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Highly Cited
1993
Highly Cited
1993
The effects of the nitric oxide (NO) donor 3-morpholino-sydnonimine (SIN-1) on the L-type Ca2+ current (ICa) were examined in… (More)
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Highly Cited
1989
Highly Cited
1989
The vasodilator and antiaggregatory properties of sydnonimines like SIN-1 are thought to be due to their marked stimulatory… (More)
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1989
1989
The antianginal effect of sydnonimines is believed to be due to the release of nitric oxide (NO) from inactive prodrugs. The… (More)
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1989
1989
Using different techniques, we measured the kinetics of nitric oxide (NO) liberation from SIN-1, the metabolite of molsidomine… (More)
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