Skip to search form
Skip to main content
Skip to account menu
Semantic Scholar
Semantic Scholar's Logo
Search 224,979,800 papers from all fields of science
Search
Sign In
Create Free Account
SIN 1C
Known as:
SIN-1C
National Institutes of Health
Create Alert
Alert
Related topics
Related topics
2 relations
Broader (2)
Acetonitriles
Morpholines
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
2015
2015
Neuroprotective effect of 3-morpholinosydnonimine against Zn²⁺-induced PC12 cell death.
J. An
,
Seong Ah Moon
,
Soo Young Hong
,
J. Kang
,
J. Seo
European Journal of Pharmacology
2015
Corpus ID: 37419635
2003
2003
Novel SIN-1 reactive intermediates modulate chloride secretion across murine airway cells.
U. Thome
,
A. Lazrak
,
+4 authors
S. Matalon
Free Radical Biology & Medicine
2003
Corpus ID: 41296978
2001
2001
Nitric oxide reduces energy supply by direct action on the respiratory chain in isolated cardiomyocytes.
T. Stumpe
,
U. Decking
,
J. Schrader
American Journal of Physiology. Heart and…
2001
Corpus ID: 7413801
To investigate the effect of nitric oxide (NO) on cardiac energy metabolism, isolated cardiomyocytes of Wistar rats were…
Expand
1998
1998
Protection of isolated cardiomyocytes against reoxygenation-induced hypercontracture by SIN-1C
B. Siegmund
,
C. Balser
,
Y. Ladilov
,
H. Piper
Basic Research in Cardiology
1998
Corpus ID: 21893205
Abstract Previous studies have shown that SIN-1C (N-morpholinoiminoacetonitrile) can protect ischemic-reperfused myocardium. The…
Expand
Review
1998
Review
1998
[Molsidomine: importance in treatment of circulation disorders].
M. Kmieć
,
W. Ochmański
Przeglad lekarski
1998
Corpus ID: 19258667
Molsidomine, coronary drug which acts similar to organic nitrates, belongs to the drug class of sydnones . SIN-1A metabolite of…
Expand
1995
1995
Intracoronary SIN‐1C During Reperfusion Reduces Infarct Size in Dog
W. Schlack
,
A. Uebing
,
+5 authors
V. Thämer
Journal of Cardiovascular Pharmacology
1995
Corpus ID: 24315352
Summary Reperfusion of ischemic myocardium may aggravate the ischemic state of injury and thus augment infarct size (reperfusion…
Expand
Highly Cited
1994
Highly Cited
1994
NO donor SIN-1 protects against reoxygenation-induced cardiomyocyte injury by a dual action.
K. Schlüter
,
M. Weber
,
E. Schraven
,
H. Piper
American Journal of Physiology
1994
Corpus ID: 20192325
It was investigated whether morpholinosydnonimine (SIN-1), which spontaneously decomposes into NO and 3…
Expand
1994
1994
Regulatory role of nitric oxide in the IL-4-induced IgE production by normal human peripheral blood mononuclear cells.
N. Paul‐eugene
,
J. Kolb
,
C. Damais
,
K. Yamaoka
,
B. Dugas
Lymphokine and cytokine research
1994
Corpus ID: 9557204
An in vitro study was performed in order to assess a possible regulatory role of nitric oxide (NO), a short-lived biologic…
Expand
By clicking accept or continuing to use the site, you agree to the terms outlined in our
Privacy Policy
(opens in a new tab)
,
Terms of Service
(opens in a new tab)
, and
Dataset License
(opens in a new tab)
ACCEPT & CONTINUE