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Recentin

 
National Institutes of Health

Papers overview

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Highly Cited
2012
Highly Cited
2012
PURPOSE Cediranib is a highly potent inhibitor of vascular endothelial growth factor (VEGF) signaling with activity against all… Expand
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2010
2010
VEGFR and c-Kit signaling pathways may contribute to the pathophysiology of acute myeloid leukemia (AML). Thirty-five patients… Expand
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2009
2009
PurposeCediranib (recentin, AZD2171) is an oral small-molecule multiple receptor tyrosine kinases inhibitor. Here we investigate… Expand
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2009
2009
PurposeTo evaluate safety and tolerability of cediranib, a highly potent and selective vascular endothelial growth factor… Expand
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2009
2009
Cediranib (RECENTIN, AZD2171) is a highly potent inhibitor of the tyrosine kinase activity associated with all three vascular… Expand
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2009
2009
Brain metastasis has become an increasing cause of morbidity and mortality in cancer patients as the treatment of systemic… Expand
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Review
2009
Review
2009
Angiogenesis, the process whereby tumors develop new blood vessels to facilitate growth and metastasis, is a pivotal event in… Expand
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2008
2008
PURPOSE Inhibition of vascular endothelial growth factor-A (VEGF) signaling is a key therapeutic approach in oncology given the… Expand
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Highly Cited
2008
Highly Cited
2008
Solid tumors express a range of factors required to sustain their growth and promote their dissemination. Among these are… Expand
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2008
2008
A number of pre-clinical studies have suggested that blocking vascular endothelial growth factor (VEGF) signalling can be… Expand
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