PSEUDOHYPOALDOSTERONISM, TYPE IID

Known as: FAMILIAL HYPERKALEMIC HYPERTENSION, FHHT, PHA2D 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

2007-2018
012320072018

Papers overview

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Review
2018
Review
2018
Autosomal dominant mutations in Cullin3 (Cul3) cause the most severe form of Familial Hyperkalemic Hypertension (FHHt). Cul3… (More)
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2017
2017
Aberrant activation of with no lysine (WNK) kinases causes familial hyperkalemic hypertension (FHHt). Thiazide diuretics treat… (More)
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2016
2016
Mutations in WNK1 and WNK4, and in components of the Cullin-Ring Ligase system, kelch-like 3 (KLHL3) and Cullin 3 (CUL3), can… (More)
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2014
2014
The with-no-lysine (K) kinases, WNK1 and WNK4, are key regulators of blood pressure. Their mutations lead to familial… (More)
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2014
2014
Familial hyperkalemic hypertension (FHHt) is a monogenic disease resulting from mutations in genes encoding WNK kinases, the… (More)
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2013
2013
Large-conductance, Ca(2+)-activated K(+) channels, commonly referred to as BK channels, have a major role in flow-induced K… (More)
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Review
2013
Review
2013
The DCT (distal convoluted tubule) is the site of microregulation of water reabsorption and ion handling in the kidneys, which is… (More)
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2012
2012
Renal excretion of Na(+) and K(+) must be regulated independently within the distal nephron, but is complicated by the fact that… (More)
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Review
2012
Review
2012
The distal nephron, which is the site of the micro-regulation of water absorption and ion handling in the kidneys, is under the… (More)
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Highly Cited
2007
Highly Cited
2007
The pathogenesis of essential hypertension remains unknown, but thiazide diuretics are frequently recommended as first-line… (More)
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