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NU 7026

Known as: NU-7026, NU7026 
National Institutes of Health

Papers overview

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2016
2016
Identification of DNA-PKcs as a primary resistance factor of salinomycin in osteosarcoma cells
Malignant osteosarcoma (OS) is still a deadly disease for many affected patients. The search for the novel anti-OS agent is… 
2015
2015
DNA-PKcs interference sensitizes colorectal cancer cells to a mTOR kinase inhibitor WAY-600.
2015
2015
The influence of ATM, ATR, DNA-PK inhibitors on the cytotoxic and genotoxic effects of dibenzo[def,p]chrysene on human hepatocellular cancer cell line HepG2.
2014
2014
Activation of DNA damage repair pathways in response to nitrogen mustard-induced DNA damage and toxicity in skin keratinocytes.
2012
2012
The catalytic subunit of DNA‐dependent protein kinase is downstream of ATM and feeds forward oxidative stress in selenium‐induced senescence response
2011
2011
Error-prone nonhomologous end joining repair operates in human pluripotent stem cells during late G2
Genome stability of human embryonic stem cells (hESC) is an important issue because even minor genetic alterations can negatively… 
2010
2010
DNA-PK, ATM and MDR proteins inhibitors in overcoming fludarabine resistance in CLL cells.
AIM To perform the comparative study of the effects of DNA-dependent protein kinase (DNA-PK) inhibitors vanillin and NU7026… 
Highly Cited
2009
Highly Cited
2009
DNA-Dependent Protein Kinase (DNA-PK)–Dependent Cisplatin-Induced Loss of Nucleolar Facilitator of Chromatin Transcription (FACT) and Regulation of Cisplatin Sensitivity by DNA-PK and FACT
Both the Ku subunit of the DNA-dependent protein kinase (DNA-PK) and the facilitator of chromatin transcription (FACT) complex… 
Highly Cited
2007
Highly Cited
2007
Chlorambucil Cytotoxicity in Malignant B Lymphocytes Is Synergistically Increased by 2-(Morpholin-4-yl)-benzo[h]chomen-4-one (NU7026)-Mediated Inhibition of DNA Double-Strand Break Repair via…
Chlorambucil (CLB) treatment is used in chronic lymphocytic leukemia (CLL) but resistance to CLB develops in association with… 
Highly Cited
2005
Highly Cited
2005
Human chronic lymphocytic leukemia B cells can escape DNA damage-induced apoptosis through the nonhomologous end-joining DNA repair pathway.
Nonhomologous end-joining (NHEJ) DNA factors maintain genomic stability through their DNA double-strand break (DSB) repair and… 
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