Skip to search formSkip to main content
You are currently offline. Some features of the site may not work correctly.

MYD88 wt Allele

Known as: Myeloid Differentiation Primary Response 88 wt Allele, Myeloid Differentiation Primary Response Gene (88), Myeloid Differentiation Primary Response Gene 88 
Human MYD88 wild-type allele is located in the vicinity of 3p22 and is approximately 5 kb in length. This allele, which encodes myeloid… Expand
National Institutes of Health

Related topics

Related topics

6 relations
3p22ImmunityMYD88 DeficiencySignal Transduction
Expand

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2018
2018
Nuclear PTEN enhances the maturation of a microRNA regulon to limit MyD88-dependent susceptibility to sepsis
Nuclear localization of the phosphatase PTEN prevents cytokine storm–mediated organ injury during sepsis. PTEN and sepsis The… Expand
2015
2015
Amyloid-beta mediates the receptor of advanced glycation end product-induced pro-inflammatory response via toll-like receptor 4 signaling pathway in retinal ganglion cell line RGC-5.
Patients with diabetes mellitus have an increased risk of developing Alzheimer's disease. Amyloid-β, a product of amyloid… Expand
2014
2014
Gut commensal bacteria and regional Wnt gene expression in the proximal versus distal colon.
Regional expression of Wingless/Int (Wnt) genes plays a central role in regulating intestinal development and homeostasis… Expand
  • figure 1
  • figure 2
  • figure 3
2014
2014
Inhibition of Arthritis in the Lewis Rat by Apolipoprotein A-I and Reconstituted High-Density Lipoproteins
  • B. Wu, K. Ong, +4 authors K. Rye
  • Arteriosclerosis, thrombosis, and vascular…
  • 2014
    • Corpus ID: 386280
Objective— This study questions whether high-density lipoproteins (HDLs) and apolipoprotein A-I inhibit joint inflammation in… Expand
  • figure 1
  • figure 2
  • figure 3
  • figure 4
  • figure 5
2013
2013
Development of high-resolution melting analysis for the detection of the MYD88 L265P mutation.
OBJECTIVE Recurrent L265P mutation of myeloid differentiation primary response gene 88 (MYD88) has been identified in a… Expand
  • figure 1
  • figure 2
Highly Cited
2012
Highly Cited
2012
Inductive expression of toll-like receptor 5 (TLR5) and associated downstream signaling molecules following ligand exposure and bacterial infection in the Indian major carp, mrigal (Cirrhinus mrigala…
Toll-like receptors (TLRs) are one of the key components of innate immunity. Among various types of TLRs, TLR5 is involved in… Expand
2012
2012
Hypoxia attenuates inflammatory mediators production induced by Acanthamoeba via Toll-like receptor 4 signaling in human corneal epithelial cells.
  • H. Pan, X. Wu
  • Biochemical and biophysical research…
  • 2012
    • Corpus ID: 44861640
Acanthamoeba keratitis (AK) is a vision-threatening corneal infection that is intimately associated with contact lens use which… Expand
  • table 1
  • figure 1
  • figure 2
  • figure 3
  • figure 4
2011
2011
Instant blood-mediated inflammatory reaction during islet transplantation: the role of Toll-like receptors signaling pathways.
The instant blood-mediated inflammatory reaction (IBMIR) leads to massive destruction of transplanted islets. Islet isolation and… Expand
  • figure 1
  • figure 2
Highly Cited
2008
Highly Cited
2008
Myeloid differentiation primary response gene 88 is required for the resolution of otitis media.
BACKGROUND Signaling defects in the Toll-like receptor (TLR) pathway, such as interleukin-1 receptor-associated kinase 4… Expand
  • figure 1
  • figure 2
  • figure 3
  • figure 4
  • table 1
Highly Cited
2006
Highly Cited
2006
Induced expression of pattern recognition receptors in cumulus oocyte complexes: novel evidence for innate immune-like functions during ovulation.
Ovulation is the complex, inflammatory-like process by which the cumulus oocyte complex (COC) is released from a mature… Expand
  • figure 1
  • figure 3
  • figure 2
  • figure 4
  • figure 5
By clicking accept or continuing to use the site, you agree to the terms outlined in our Privacy Policy, Terms of Service, and Dataset License
ACCEPT & CONTINUE