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MAP4K2 gene
Known as:
BL44
, MITOGEN-ACTIVATED PROTEIN KINASE KINASE KINASE KINASE 2
, GERMINAL CENTER KINASE
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National Institutes of Health
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Related topics
1 relation
germinal center kinases
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2017
Highly Cited
2017
Targeted next-generation sequencing reveals MODY in up to 6.5% of antibody-negative diabetes cases listed in the Norwegian Childhood Diabetes Registry
B. Johansson
,
Henrik Irgens
,
+10 authors
P. Njølstad
Diabetologia
2017
Corpus ID: 8973746
Aims/hypothesisMODY can be wrongly diagnosed as type 1 diabetes in children. We aimed to find the prevalence of MODY in a…
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Review
2016
Review
2016
Perspective on the Genetics and Diagnosis of Congenital Hyperinsulinism Disorders.
C. Stanley
Journal of Clinical Endocrinology and Metabolism
2016
Corpus ID: 2661274
CONTEXT Congenital hyperinsulinism (HI) is the most common cause of hypoglycemia in children. The risk of permanent brain injury…
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Highly Cited
2013
Highly Cited
2013
Genotype and phenotype correlations in 417 children with congenital hyperinsulinism.
K. Snider
,
Susan A. Becker
,
+7 authors
A. Ganguly
Journal of Clinical Endocrinology and Metabolism
2013
Corpus ID: 22272204
CONTEXT Hypoglycemia due to congenital hyperinsulinism (HI) is caused by mutations in 9 genes. OBJECTIVE Our objective was to…
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Highly Cited
2012
Highly Cited
2012
Comprehensive molecular analysis of Japanese patients with pediatric‐onset MODY‐type diabetes mellitus
T. Yorifuji
,
R. Fujimaru
,
+9 authors
T. Urakami
Pediatric Diabetes
2012
Corpus ID: 21337182
Yorifuji T, Fujimaru R, Hosokawa Y, Tamagawa N, Shiozaki M, Aizu K, Jinno K, Maruo Y, Nagasaka H, Tajima T, Kobayashi K, Urakami…
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Highly Cited
2012
Highly Cited
2012
Incidence, genetics, and clinical phenotype of permanent neonatal diabetes mellitus in northwest Saudi Arabia
A. Habeb
,
Mohamed S Al-Magamsi
,
+4 authors
S. Ellard
Pediatric Diabetes
2012
Corpus ID: 45043646
Habeb AM, Al‐Magamsi MSF, Eid IM, Ali MI, Hattersley AT, Hussain K, Ellard S. Incidence, genetics, and clinical phenotype of…
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Highly Cited
2011
Highly Cited
2011
Association of Genetic Loci With Glucose Levels in Childhood and Adolescence
Adam Barker
,
S. Sharp
,
+31 authors
C. Langenberg
Diabetes
2011
Corpus ID: 7796929
OBJECTIVE To investigate whether associations of common genetic variants recently identified for fasting glucose or insulin…
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Highly Cited
2008
Highly Cited
2008
Impact of Common Type 2 Diabetes Risk Polymorphisms in the DESIR Prospective Study
M. Vaxillaire
,
J. Veslot
,
+9 authors
P. Froguel
Diabetes
2008
Corpus ID: 10405185
OBJECTIVE— The emerging picture of type 2 diabetes genetics involves differently assembled gene variants, each modestly…
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Highly Cited
2005
Highly Cited
2005
Polymorphisms in the SLC2A2 (GLUT2) gene are associated with the conversion from impaired glucose tolerance to type 2 diabetes: the Finnish Diabetes Prevention Study.
O. Laukkanen
,
J. Lindström
,
+7 authors
M. Laakso
Diabetes
2005
Corpus ID: 45463873
Impaired insulin secretion is a fundamental defect in type 2 diabetes. The aim of this study was to investigate whether single…
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Review
2001
Review
2001
The Ste20 group kinases as regulators of MAP kinase cascades.
Ippeita Dan
,
Norinobu M. Watanabe
,
A. Kusumi
Trends in Cell Biology
2001
Corpus ID: 32220148
Highly Cited
1998
Highly Cited
1998
Tumor Necrosis Factor Signaling to Stress-activated Protein Kinase (SAPK)/Jun NH2-terminal Kinase (JNK) and p38
T. Yuasa
,
S. Ohno
,
J. Kehrl
,
J. Kyriakis
Journal of Biological Chemistry
1998
Corpus ID: 23642317
Tumor necrosis factor (TNF) elicits a diverse array of inflammatory responses through engagement of its type-1 receptor (TNFR1…
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