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KU 14R

Known as: KU-14R, KU14R 
National Institutes of Health

Related topics

Related topics

2 relations

Broader (2)

BenzofuransImidazoles

Papers overview

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2011
2011
Imidazoleacetic acid-ribotide induces depression of synaptic responses in hippocampus through activation of imidazoline receptors.
Imidazole-4-acetic acid-ribotide (IAA-RP), an endogenous agonist at imidazoline receptors (I-Rs), is a putative neurotransmitter… 
2005
2005
Essential role of the imidazoline moiety in the insulinotropic effect but not the KATP channel-blocking effect of imidazolines; a comparison of the effects of efaroxan and its imidazole analogue…
Aims/hypothesisImidazolines are a class of investigational antidiabetic drugs. It is still unclear whether the imidazoline ring… 
2003
2003
Evidence for Nonadrenoceptor Responses to Imidazoline Derivatives in the Porcine Isolated Rectal Artery
Abstract: High concentrations of phentolamine, efaroxan, and idazoxan were found to produce nonadrenoceptor contractions of the… 
2002
2002
Effects of the imidazoline ligands efaroxan and KU14R on blood glucose homeostasis in the mouse.
2001
2001
Characterization of a KATP channel-independent pathway involved in potentiation of insulin secretion by efaroxan.
Efaroxan, like several other imidazoline reagents, elicits a glucose-dependent increase in insulin secretion from pancreatic beta… 
2000
2000
Stimulation of insulin secretion in clonal BRIN-BD11 cells by the imidazoline derivatives KU14r and RX801080.
The imidazoline derivatives KU14R and RX801080 have each been reported to antagonize imidazoline-stimulated insulin secretion… 
1999
1999
Multiple effector pathways regulate the insulin secretory response to the imidazoline RX871024 in isolated rat pancreatic islets
When isolated rat islets were cultured for 18 h prior to use, the putative imidazoline binding site ligand, RX871024 caused a… 
1998
1998
Characterisation of new efaroxan derivatives for use in purification of imidazoline-binding sites.
1997
1997
Evidence that the ability of imidazoline compounds to stimulate insulin secretion is not due to interaction with sigma receptors.
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