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KT-7515

Known as: KT 7515, KT7515 
 
National Institutes of Health

Papers overview

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Highly Cited
2002
Highly Cited
2002
Nerve growth factor (NGF) deprivation triggers metabolic changes in sympathetic neurons that precede cell death. Here, we… Expand
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2002
2002
Cerebellar granule neurons grown in high potassium undergo rapid apoptosis when switched to medium containing 5 mm potassium, a… Expand
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2002
2002
Overexpression of gp120, the major coat protein of the HIV-1 virus, in central glial cells, or treatment of neurons with gp120 in… Expand
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Highly Cited
2001
Highly Cited
2001
CEP-1347 (KT7515) promotes neuronal survival at dosages that inhibit activation of the c-Jun amino-terminal kinases (JNKs) in… Expand
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Highly Cited
2001
Highly Cited
2001
beta-Amyloid (A beta) has been strongly implicated in the pathophysiology of Alzheimer's disease (AD), but the means by which the… Expand
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2001
2001
BACKGROUND Once paracrine growth factors are secreted by androgen receptor expressing prostatic stromal cells, they diffuse… Expand
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2000
2000
Previous studies have implicated stress-activated protein kinases (SAPKs) in aberrant phosphorylation of the high-molecular-mass… Expand
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Highly Cited
1999
Highly Cited
1999
The c-Jun N-terminal kinase signaling cascade appears to play a role in some cases of cell death, including neuronal apoptosis… Expand
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Highly Cited
1998
Highly Cited
1998
Neurons undergoing apoptosis can be rescued by trophic factors that simultaneously increase the activity of extracellular signal… Expand
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1998
1998
CEP-1347, also known as KT7515, a derivative of a natural product indolocarbazole, inhibited motor neuronal death in vitro… Expand
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