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Hypocalciuria
Known as:
Low urine calcium levels
An abnormally decreased calcium concentration in the urine (FMA:12274). [HPO:probinson]
National Institutes of Health
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Related topics
Related topics
7 relations
Gitelman Syndrome
HYPOCALCIURIC HYPERCALCEMIA, FAMILIAL, TYPE III
Hypocalcemia
Hypocalciuric hypercalcemia, familial, type 1
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Broader (1)
calcium deficiency
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2010
Highly Cited
2010
Protective effect of the hydro-alcoholic extract of Rubia cordifolia roots against ethylene glycol induced urolithiasis in rats.
K. Divakar
,
A. Pawar
,
S. Chandrasekhar
,
S. Dighe
,
G. Divakar
Food and Chemical Toxicology
2010
Corpus ID: 24062695
Highly Cited
2007
Highly Cited
2007
Decreased fractional urinary calcium excretion and serum 1,25-dihydroxyvitamin D and IGF-I levels in preeclampsia
A. Halhali
,
L. Díaz
,
E. Avila
,
A. Ariza
,
M. Garabédian
,
F. Larrea
Journal of Steroid Biochemistry and Molecular…
2007
Corpus ID: 31097210
Highly Cited
2003
Highly Cited
2003
Thiazide-induced hypocalciuria is accompanied by a decreased expression of Ca2+ transport proteins in kidney.
Tom Nijenhuis
,
J. Hoenderop
,
J. Loffing
,
A. W. van der Kemp
,
C. van Os
,
R. Bindels
Kidney International
2003
Corpus ID: 26768792
INTRODUCTION Thiazide diuretics have the unique characteristic of increasing renal Na+ excretion, while decreasing Ca2+ excretion…
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Highly Cited
2003
Highly Cited
2003
A novel mutation in the chloride channel gene, CLCNKB, as a cause of Gitelman and Bartter syndromes.
I. Zelikovic
,
R. Szargel
,
+4 authors
F. Nakhoul
Kidney International
2003
Corpus ID: 25405145
BACKGROUND Gitelman syndrome (GS) and Bartter syndrome (BS) are hereditary hypokalemic tubulopathies with distinct phenotypic…
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Review
2003
Review
2003
(Patho)physiological implications of the novel epithelial Ca2+ channels TRPV5 and TRPV6
Tom Nijenhuis
,
J. Hoenderop
,
B. Nilius
,
R. Bindels
Pflügers Archiv
2003
Corpus ID: 21297465
The epithelial Ca2+ channels TRPV5 and TRPV6 constitute the apical Ca2+ entry mechanism in active Ca2+ (re)absorption. These two…
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Highly Cited
2002
Highly Cited
2002
Clinical presentation of genetically defined patients with hypokalemic salt-losing tubulopathies.
M. Peters
,
N. Jeck
,
+5 authors
H. Seyberth
American Journal of Medicine
2002
Corpus ID: 24018513
Highly Cited
2000
Highly Cited
2000
Mutations in the chloride channel gene CLCNKB as a cause of classic Bartter syndrome.
M. Konrad
,
M. Vollmer
,
+12 authors
F. Hildebrandt
Journal of the American Society of Nephrology
2000
Corpus ID: 2104532
ABSTRACT.: Inherited hypokalemic renal tubulopathies are differentiated into at least three clinical subtypes: (1) the Gitelman…
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Highly Cited
1995
Highly Cited
1995
Possible discrimination of Gitelman's syndrome from Bartter's syndrome by renal clearance study: report of two cases.
T. Tsukamoto
,
Tatsuya Kobayashi
,
Kunihiko Kawamoto
,
Masaaki Fukase
,
Kazuo Chihara
American Journal of Kidney Diseases
1995
Corpus ID: 45322206
Highly Cited
1992
Highly Cited
1992
Use of calcium excretion values to distinguish two forms of primary renal tubular hypokalemic alkalosis: Bartter and Gitelman syndromes.
A. Bettinelli
,
M. Bianchetti
,
+13 authors
P. Vitucci
Jornal de Pediatria
1992
Corpus ID: 22879173
Highly Cited
1969
Highly Cited
1969
Chlorothiazide-induced hypercalcemia in juvenile osteoporosis and hyperparathyroidism.
A. Parfitt
New England Journal of Medicine
1969
Corpus ID: 43341688
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