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HOE 140

Known as: HOE-140, Hoechst-140, Hoechst 140 
 
National Institutes of Health

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Highly Cited
2007
Highly Cited
2007
OBJECTIVE Postconditioning (PostC) maneuvers allow post-ischemic accumulation of autacoids, which trigger protection. We tested… Expand
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Highly Cited
2004
Highly Cited
2004
Background—Angiotensin (Ang) II type 2 (AT2) receptor stimulation results in coronary vasodilation in the rat heart. In contrast… Expand
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Highly Cited
2003
Highly Cited
2003
Abstract—We have previously demonstrated that stimulation of the angiotensin (Ang) II type 2 receptor in vascular smooth muscle… Expand
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Highly Cited
2002
Highly Cited
2002
Heterozygosity for C1 inhibitor (C1INH) deficiency results in hereditary angioedema. Disruption of the C1INH gene by gene… Expand
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Highly Cited
2002
Highly Cited
2002
Objective: Myocardial protection can be achieved by brief ischemia-reperfusion of remote organs, a phenomenon described as remote… Expand
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Highly Cited
2002
Highly Cited
2002
Improvement of insulin resistance by ACE inhibitors has been suggested; however, this mechanism has not been proved. We… Expand
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Highly Cited
1998
Highly Cited
1998
OBJECTIVE We studied the effect of the angiotensin II type 1 (AT1)-receptor antagonist candesartan on infarct size resulting from… Expand
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Highly Cited
1997
Highly Cited
1997
We have shown previously that angiotensin-converting enzyme (ACE) inhibitors prevent coronary vascular remodeling (medial… Expand
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Highly Cited
1995
Highly Cited
1995
The kallikrein-kinin system is involved in the inflammatory process, in blood pressure regulation, and in renal homeostasis. The… Expand
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Highly Cited
1992
Highly Cited
1992
The effect of peripheral and intrathecal administration of the bradykinin B2 receptor antagonist HOE140 and the non-steroidal… Expand
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