HMR 1766

Known as: HMR-1766, HMR1766 
 
National Institutes of Health

Topic mentions per year

Topic mentions per year

2004-2017
012320042017

Papers overview

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2011
2011
Oxidation and loss of heme in soluble guanylyl/guanylate cyclase (sGC), the nitric oxide receptor, is thought to be a major… (More)
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2010
2010
OBJECTIVE We investigated the mechanisms underlying vascular endothelial and contractile dysfunction in diabetes as well as the… (More)
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2009
2009
Severe pulmonary hypertension (PH) is a disabling disease with high mortality, characterized by pulmonary vascular remodeling and… (More)
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2009
2009
AIMS Ischaemia-reperfusion provokes barrier failure of the coronary microvasculature, impeding functional recovery of the heart… (More)
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2008
2008
Many vascular diseases are characterized by increased levels of ROS that destroy the biological activity of nitric oxide and… (More)
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2008
2008
Abbreviations: 3-isobutyl-1-methylxanthine (IBMX); 5(6) carboxy-2 ́,7 ́dichlorofluorescein diacetate (carboxy-DCFDA), BAY 58-2667… (More)
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2007
2007
Nitric oxide formation is impaired in chronic renal failure. The renoprotective effects of a nonhypotensive dose of HMR1766, a… (More)
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2007
2007
HMR1766 is a new nitric oxide (NO)-independent activator of soluble guanylyl cyclase (sGC) in development for the treatment of… (More)
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2006
2006
The heme-enzyme soluble guanylyl cyclase (sGC) is an ubiquitous NO receptor, which mediates NO downstream signaling by the… (More)
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2006
2006
OBJECTIVE Platelet activation significantly contributes to cardiovascular morbidity and mortality in diabetes. An association… (More)
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