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GTS-21

Known as: GTS 21 
National Institutes of Health

Papers overview

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Highly Cited
2011
Highly Cited
2011
The vagus nerve can reflexively attenuate the innate immune response via binding of the vagal neurotransmitter acetylcholine (ACh… 
Highly Cited
2009
Highly Cited
2009
The cholinergic antiinflammatory pathway modulates Inflammatory cytokine production through a mechanism dependent on the vagus… 
Highly Cited
2007
Highly Cited
2007
Objective:Tumor necrosis factor and high mobility group box 1 are critical cytokine mediators of inflammation. The efferent vagus… 
Highly Cited
2006
Highly Cited
2006
BACKGROUND & AIMS The nervous system, through the vagus nerve, controls inflammation by decreasing the release of tumor necrosis… 
Highly Cited
2003
Highly Cited
2003
This study was designed to determine the safety, tolerability, pharmacokinetics and effects on cognitive function of GTS-21 in… 
Highly Cited
2002
Highly Cited
2002
Pharmacological studies of α7 nicotinic acetylcholine receptors are confounded by the fact that rapid desensitization to high… 
Highly Cited
1998
Highly Cited
1998
The neuroprotective effects of GTS-21 [3-(2,4-dimethoxybenzylidene)-anabaseine dihydrochloride] were studied and compared with… 
Highly Cited
1998