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GS967

National Institutes of Health

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2018
2018
ICaL and Ito mediate rate-dependent repolarization in rabbit atrial myocytes
Rate-dependent repolarization (RDR) of action potential (AP) in cardiomyocyte plays a critical role in the genesis of arrhythmias… 
2017
2017
ICaL and Ito mediate rate-dependent repolarization in rabbit atrial myocytes
Rate-dependent repolarization (RDR) of action potential (AP) in cardiomyocyte plays a critical role in the genesis of arrhythmias… 
2016
2016
Pre- and Delayed Treatments With Ranolazine Ameliorate Ventricular Arrhythmias and Nav1.5 Downregulation in Ischemic/Reperfused Rat Hearts
Abstract: Enhanced late sodium current (late INa) and intracellular Nav1.5 redistribution contribute to ischemia/reperfusion (I/R… 
2015
2015
THE LATE SODIUM CURRENT INHIBITOR GS967 SUPPRESSES SPONTANEOUS ATRIAL FIBRILLATION IN AN INTACT PORCINE MODEL
2015
2015
SELECTIVE LATE INA INHIBITION BY GS967 EXERTS POTENT SUPPRESSION OF CATECHOLAMINE-INDUCED VENTRICULAR TACHYCARDIA
2014
2014
Selective Inhibition of the Late Sodium Current has No Adverse Effect on Electrophysiological or Contractile Function of the Normal Heart
Abstract: Inhibition of cardiac late Na+ current (INa,L) decreases sodium-dependent calcium overload in diseased hearts. Because… 
2014
2014
Abstract 14019: Electrophysiological Effects of the Late Sodium Current Inhibitor GS967 in Scn5a-1798insD Mouse and Human SCN5A-1795insD iPSC-derived Cardiomyocytes
Background and Aim: Selective inhibition of cardiac late sodium current (INa,L) is an emerging target in the treatment of… 
2014
2014
Rebuttal from Marc Pourrier, Sarah Williams, Donald McAfee, Luiz Belardinelli and David Fedida
Papp et al. (2013) raise a number of salient points in disputing the importance of INa,L in diastolic dysfunction in HFpEF. First… 
2013
2013
Increased Nav 1.5 channel late current underlies prolongation of action potential duration of ventricular myocytes from aged guinea pigs
2012
2012
Late Sodium Current Is an Intrinsic Regulator of Cardiac Repolarization—A Quantitative Assessment
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