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FM19G11

National Institutes of Health

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Broader (1)

Benzamides

Papers overview

Semantic Scholar uses AI to extract papers important to this topic.
2019
2019
Possible Contribution of Inflammation-Associated Hypoxia to Increased K2P5.1 K+ Channel Expression in CD4+ T Cells of the Mouse Model for Inflammatory Bowel Disease
Previous studies have reported the up-regulation of the two-pore domain K+ channel K2P5.1 in the CD4+ T cells of patients with… 
2018
2018
FM19G11 and Ependymal Progenitor/Stem Cell Combinatory Treatment Enhances Neuronal Preservation and Oligodendrogenesis after Severe Spinal Cord Injury
Spinal cord injury (SCI) suffers from a lack of effective therapeutic strategies. We have previously shown that individual… 
2018
2018
FM19G11 inhibits O6‐methylguanine DNA‐methyltransferase expression under both hypoxic and normoxic conditions
FM19G11 is a small molecular agent that inhibits hypoxia‐inducible factor‐1‐alpha (HIF‐1α) and other signaling pathways. In this… 
2015
2015
Methacrylate‐endcapped caprolactone and FM19G11 provide a proper niche for spinal cord‐derived neural cells
Spinal cord injury (SCI) is a cause of paralysis. Although some strategies have been proposed to palliate the severity of this… 
2013
2013
Inhibition of mTOR and HIF pathways diminishes chondro-osteogenesis and cell proliferation in chondroblastoma
Chondroblastoma (CBL) is a benign bone tumor occurring mostly in teenagers. Despite this, CBL can recur and metastasize after… 
2012
2012
FM19G11 Favors Spinal Cord Injury Regeneration and Stem Cell Self‐Renewal by Mitochondrial Uncoupling and Glucose Metabolism Induction
Spinal cord injury is a major cause of paralysis with no currently effective therapies. Induction of self‐renewal and… 
2010
2010
FM19G11: A new modulator of HIF that links mTOR activation with the DNA damage checkpoint pathways
The network consisting of mTOR and p53 pathways is crucial to understanding a wide variety of physiological and pathological… 
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