Skip to search form
Skip to main content
Skip to account menu
Semantic Scholar
Semantic Scholar's Logo
Search 225,436,945 papers from all fields of science
Search
Sign In
Create Free Account
FHOD3 gene
Known as:
FORMIN HOMOLOGY-2 DOMAIN-CONTAINING PROTEIN 3
, KIAA1695
, FHOS2
Expand
National Institutes of Health
Create Alert
Alert
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
Review
2019
Review
2019
54 Novel formin homology 2 domain containing 3 (FHOD3) mutations associated with the pathogenesis of hypertrophic cardiomyopathy (HCM) in an irish population
J. Carron
,
C. Mcgorrian
,
J. Galvin
,
M. Gallagher
,
J. O"brien
General Poster Abstracts 2
2019
Corpus ID: 213316912
Background The genetic cause of hypertrophic cardiomyopathy (HCM) remains unexplained in a substantial proportion of cases…
Expand
2017
2017
Crucial Role of ROCK 2-Mediated Phosphorylation and Upregulation of FHOD 3 in the Pathogenesis of Angiotensin II – Induced Cardiac Hypertrophy
Qing Zhou
,
Si-si Wei
,
+9 authors
Yue-peng Wang
2017
Corpus ID: 40780416
2015
2015
Distribution of formins in cardiac muscle: FHOD1 is a component of intercalated discs and costameres.
A. Al Haj
,
A. Mazur
,
+6 authors
H. Mannherz
European Journal of Cell Biology
2015
Corpus ID: 19530154
2014
2014
Circulation: Cardiovascular Genetics 2013 Editors’ Picks
L. Wilkins
2014
Corpus ID: 80603859
Summary— Hypertrophic cardiomyopathy (HCM) is characterized by excessive thickening and enlargement of the heart muscle, causing…
Expand
2004
2004
Identification and characterization of human FHOD3 gene in silico.
Masuko Katoh
,
M. Katoh
International Journal of Molecular Medicine
2004
Corpus ID: 22904729
Formin homology proteins are actin regulators with scaffold function, which are implicated in organogenesis, normal tissue…
Expand
By clicking accept or continuing to use the site, you agree to the terms outlined in our
Privacy Policy
(opens in a new tab)
,
Terms of Service
(opens in a new tab)
, and
Dataset License
(opens in a new tab)
ACCEPT & CONTINUE