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DP 1904

Known as: DP-1904 
National Institutes of Health

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2004
2004
Down-regulation of lipoxin A4 receptor by thromboxane A2 signaling in RAW246.7 cells in vitro and bleomycin-induced lung fibrosis in vivo.
2001
2001
Thromboxane A(2) causes retarded clearance of aggregated protein in glomeruli of nephritic mice.
1999
1999
In vitro responses to antigen stimulation: comparison between human lung parenchyma resected from asthmatic patients and non-asthmatic patients.
1998
1998
Correlative alteration of thromboxane A2 with antigen-induced bronchoconstriction and the role of platelets as a source of TXA2 synthesis in guinea pigs: effect of DP-1904, an inhibitor of…
A marked and sustained bronchoconstriction after antigen challenge was produced in actively sensitised guinea pigs, and… 
1996
1996
Effect of DP-1904, a thromboxane A2 synthase inhibitor, on passive Heymann nephritis in rats.
1996
1996
Pharmacokinetic-pharmacodynamic modelling of DP-1904, a novel thromboxane synthetase inhibitor in rabbits, based on an indirect response model
SummaryA new imidazole derivative, DP-1904, produces a selective, potent and long-acting inhibition of thromboxane A2 (TXA2… 
1995
1995
Effect of DP-1904, a thromboxane A2 synthase inhibitor, administered from the autologous phase on crescentic-type anti-GBM nephritis in rats.
The antinephritic effect of DP-1904 [6-(1-imidazolylmethyl)-5,6,7,8-tetrahydronaphthalene-2-carboxylic acid hydrochloride], a… 
1994
1994
DP-1904, a specific inhibitor of thromboxane A2 synthesizing enzyme, suppresses ICAM-1 expression by stimulated vascular endothelial cells.
1992
1992
A thromboxane A2 synthetase inhibitor retards hypertensive rat diabetic nephropathy.
1991
1991
A thromboxane A2 synthase inhibitor, DP-1904, prevents rat renal injury.
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