Skip to search form
Skip to main content
Skip to account menu
Semantic Scholar
Semantic Scholar's Logo
Search 227,741,289 papers from all fields of science
Search
Sign In
Create Free Account
Chromosome 17 Short Arm
Known as:
17p
, Chromosome 17 Proximal Arm
, Chromosome 17p
Proximal (short) arm of chromosome 17
National Institutes of Health
Create Alert
Alert
Related topics
Related topics
13 relations
17p11.1
17p11.2
17p12-17p11.2
17p12-p11.2
Expand
Broader (1)
Chromosomes, Human
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
Highly Cited
2001
Highly Cited
2001
Genetic changes in colorectal carcinoma tumors with liver metastases analyzed by comparative genomic hybridization and DNA ploidy
K. Nakao
,
M. Shibusawa
,
+6 authors
K. Sasaki
Cancer
2001
Corpus ID: 12377766
Liver metastases are found in 10% of primary colorectal malignancies, and they affects the prognosis of patients with colorectal…
Expand
Highly Cited
1999
Highly Cited
1999
Cytogenetic profile of lymphoma of follicle mantle lineage: correlation with clinicobiologic features.
A. Cuneo
,
R. Bigoni
,
+11 authors
G. Castoldi
Blood
1999
Corpus ID: 38153431
Conventional chromosome analysis (CCA) and interphase fluorescence in situ hybridization (FISH) was performed in 42 patients with…
Expand
Highly Cited
1997
Highly Cited
1997
Patterns of allelic loss on chromosome 17 in sporadic breast carcinomas detected by fluorescent‐labeled microsatellite analysis
D. Niederacher
,
F. Picard
,
Claudia van Roeyen
,
H. An
,
H. Bender
,
M. W. Beckmann
Genes, Chromosomes and Cancer
1997
Corpus ID: 23265296
Loss of heterozygosity (LOH) on chromosome 17 is a frequent genetic alteration in breast cancer. To assess whether the location…
Expand
Highly Cited
1996
Highly Cited
1996
Comparative allelotype of early and advanced stage non‐small cell lung carcinomas
M. Shiseki
,
T. Kohno
,
+6 authors
J. Yokota
Genes, Chromosomes and Cancer
1996
Corpus ID: 29897829
To identify chromosomal loci of tumor suppressor genes involved in the genesis and progression of non‐small cell lung carcinoma…
Expand
Highly Cited
1995
Highly Cited
1995
p53 mutations and prognosis in bladder tumors.
T. Uchida
,
C. Wada
,
+5 authors
K. Koshiba
Journal of Urology
1995
Corpus ID: 28048086
Highly Cited
1994
Highly Cited
1994
Loss of heterozygosity and p53 gene mutations in breast cancer.
G. Deng
,
Ling-chun Chen
,
+5 authors
H. Smith
Cancer Research
1994
Corpus ID: 17894118
Immunopositivity for the p53 tumor suppressor gene product was evaluated in 133 breast cancers and compared to loss of…
Expand
Highly Cited
1990
Highly Cited
1990
Frequent loss of heterozygosity on chromosomes 6q, 11, and 17 in human ovarian carcinomas.
Je Hwan Lee
,
J. Kavanagh
,
D. Wildrick
,
J. Wharton
,
M. Blick
Cancer Research
1990
Corpus ID: 11024245
Recently, tumor-specific allele loss has been shown to be an important characteristic of some tumors. When such loss includes one…
Expand
Highly Cited
1990
Highly Cited
1990
Cytogenetics of colorectal adenocarcinomas.
M. Muleris
,
R. Salmon
,
B. Dutrillaux
Cancer Genetics and Cytogenetics
1990
Corpus ID: 45159663
Highly Cited
1989
Highly Cited
1989
Loss of heterozygosity for loci on chromosome 17p in human malignant astrocytoma.
D. Fults
,
R. Tippets
,
G. A. Thomas
,
Y. Nakamura
,
R. White
Cancer Research
1989
Corpus ID: 28494846
Loss of constitutional heterozygosity for specific chromosomal loci, when found consistently in a particular tumor type, suggests…
Expand
Highly Cited
1989
Highly Cited
1989
Localization of the mutation in an extended family with Charcot-Marie-Tooth neuropathy (HMSN I).
P. Raeymaekers
,
V. Timmerman
,
+9 authors
C. Broeckhoven
American Journal of Human Genetics
1989
Corpus ID: 12585511
Hereditary motor and sensory neuropathy type I (HMSN I) or Charcot-Marie-Tooth (CMT) disease is an autosomal dominant peripheral…
Expand
By clicking accept or continuing to use the site, you agree to the terms outlined in our
Privacy Policy
(opens in a new tab)
,
Terms of Service
(opens in a new tab)
, and
Dataset License
(opens in a new tab)
ACCEPT & CONTINUE