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Cerebral Hypoxia-Ischemia

Known as: Hypoxia-Ischemia, Cerebral, Cerebral Ischemia-Hypoxias, Ischemia-Hypoxia, Cerebral 
 
National Institutes of Health

Papers overview

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Highly Cited
2013
Highly Cited
2013
Significance We show that the selective overactivation of autophagy can cause cell death with unique morphological features… Expand
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Highly Cited
2008
Highly Cited
2008
Autophagy, an intracellular bulk degradation process of cellular constituents, plays a key role in cell homeostasis and can be… Expand
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Highly Cited
2007
Highly Cited
2007
Upon cerebral hypoxia-ischemia (HI), apoptosis-inducing factor (AIF) can move from mitochondria to nuclei, participate in… Expand
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Highly Cited
2003
Highly Cited
2003
Apoptosis-inducing factor (AIF) triggers apoptosis in a caspase-independent manner. Here we report for the first time involvement… Expand
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Highly Cited
2002
Highly Cited
2002
Detection of cerebral hypoxia–ischemia remains problematic in neonates. Near-infrared spectroscopy, a noninvasive bedside… Expand
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Highly Cited
2001
Highly Cited
2001
The relative contributions of apoptosis and necrosis in brain injury have been a matter of much debate. Caspase-3 has been… Expand
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Review
1999
Review
1999
  • S. Love
  • Brain pathology
  • 1999
  • Corpus ID: 40684569
Brain ischemia initiates a complex cascade of metabolic events, several of which involve the generation of nitrogen and oxygen… Expand
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Highly Cited
1998
Highly Cited
1998
Hypoxia inducible factors (HIFs) are heterodimeric transcription factors that regulate a number of adaptive responses to low… Expand
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Highly Cited
1997
Highly Cited
1997
The most frequently used model of neonatal cerebral hypoxia-ischemia consists of a 7-day postnatal rat model with combined common… Expand
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Highly Cited
1995
Highly Cited
1995
ABSTRACT: Severely birth-asphyxiated human infants develop delayed (“secondary”) cerebral energy failure, which carries a poor… Expand
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