Skip to search form
Skip to main content
Skip to account menu
Semantic Scholar
Semantic Scholar's Logo
Search 225,206,859 papers from all fields of science
Search
Sign In
Create Free Account
CT 2584
Known as:
CT-2584
, CT2584
, CT2584 HMS
A substance that is being studied in the treatment of cancer. It may prevent the growth of blood vessels from surrounding tissue into a solid tumor.
National Institutes of Health
Create Alert
Alert
Related topics
Related topics
2 relations
Narrower (1)
Apra
Broader (1)
Xanthines
Papers overview
Semantic Scholar uses AI to extract papers important to this topic.
2001
2001
CT-2584 (Cell Therapeutics).
R. Bonfil
Current opinion in investigational drugs
2001
Corpus ID: 22139733
CT-2584, an anticancer agent that inhibits phospholipid signaling, is under development by Cell Therapeutics Inc (CTI) as a…
Expand
2001
2001
CT-2584 (Cell Therapeutics).
Bonfil Rd
2001
Corpus ID: 81498599
1999
1999
Development and validation of a sensitive solid-phase extraction and high-performance liquid chromatography assay for the novel antitumour agent CT2584 in human plasma.
Parveen Khan
,
Sayara Abbas
,
Sue Cheeseman
,
Malcolm R Ranson
,
Alan T. McGown
Journal of Chromatography B: Biomedical Sciences…
1999
Corpus ID: 33532491
Highly Cited
1997
Highly Cited
1997
Endogenous natural killer enhancing factor-B increases cellular resistance to oxidative stresses.
H. Shau
,
A. Kim
,
+5 authors
D. Paglia
Free Radical Biology & Medicine
1997
Corpus ID: 41336614
1997
1997
Effect of a xanthine analog on human hepatocellular carcinoma cells (Alexander) in culture and in xenografts in SCID mice
L. Marucci
,
L. Varticovski
,
I. Arias
Hepatology
1997
Corpus ID: 19215709
Hepatocellular carcinoma (HCC) frequently overexpresses the MDR1 gene and is resistant to drugs transported by the multidrug…
Expand
By clicking accept or continuing to use the site, you agree to the terms outlined in our
Privacy Policy
(opens in a new tab)
,
Terms of Service
(opens in a new tab)
, and
Dataset License
(opens in a new tab)
ACCEPT & CONTINUE