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CL 387785

Known as: CL-387,785, CL-387785 
National Institutes of Health

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1 relation

Broader (1)

Quinazolines

Papers overview

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2018
2018
Quantitative Measurement of γ-Secretase-mediated Amyloid Precursor Protein and Notch Cleavage in Cell-based Luciferase Reporter Assay Platforms.
We have developed a pair of cell-based reporter gene assays to quantitatively measure γ-secretase cleavage of distinct substrates… 
2015
2015
EGFR participates downstream of ERα in estradiol-17β-d-glucuronide-induced impairment of Abcc2 function in isolated rat hepatocyte couplets
Estradiol-17β-d-glucuronide (E17G) induces acute endocytic internalization of canalicular transporters, including multidrug… 
2013
2013
Neuroendocrine Differentiation in Acquired Resistance to Epidermal Growth Factor Receptor Tyrosine Kinase Inhibitor
Background Small cell lung cancer (SCLC) transformation during epidermal growth factor receptor tyrosine kinase inhibitor (EGFR… 
2012
2012
Inhibition of the mTOR/S6K signal is necessary to enhance fluorouracil-induced apoptosis in gastric cancer cells with HER2 amplification.
The purpose of this study was to explore the effect of trastuzumab in enhancing the activity of chemotherapeutic agents and the… 
2012
2012
Abstract 4832: Novel EGFR mutations that cause drug resistance to irreversible pyrimidine but not quinazoline based EGFR inhibitors
Background: Oncogenic EGFR T790M causes drug resistance to quinazoline based EGFR kinase inhibitors by increasing ATP affinity… 
2005
2005
Gefitinib resistance caused by a secondary mutation of the epidermal growth factor receptor
2637 Mutations of the epidermal growth factor receptor gene conferring sensitivity to anilinoquinazoline EGFR inhibitors have… 
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