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CL 387785

Known as: CL-387,785, CL-387785 
 
National Institutes of Health

Papers overview

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2015
2015
Estradiol-17β-d-glucuronide (E17G) induces acute endocytic internalization of canalicular transporters, including multidrug… Expand
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2013
2013
Background Small cell lung cancer (SCLC) transformation during epidermal growth factor receptor tyrosine kinase inhibitor (EGFR… Expand
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2012
2012
Epidermal growth factor receptor (EGFR) mutational status, activation of downstream signaling, and effective apoptotic cascade… Expand
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2012
2012
The purpose of this study was to explore the effect of trastuzumab in enhancing the activity of chemotherapeutic agents and the… Expand
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2012
2012
Background: Oncogenic EGFR T790M causes drug resistance to quinazoline based EGFR kinase inhibitors by increasing ATP affinity… Expand
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Highly Cited
2011
Highly Cited
2011
BACKGROUND Epithelial-to-mesenchymal transition (EMT), which was related with an acquired resistance to gefitinib, was found in… Expand
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2011
2011
δ-Opioid receptor (DOR)-induced activation of extracellular signal-regulated kinases 1 and 2 (ERK1/2) is mediated by the… Expand
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Highly Cited
2008
Highly Cited
2008
The epidermal growth factor receptor (EGFR) secondary kinase domain T790M non-small cell lung cancer (NSCLC) mutation enhances… Expand
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Highly Cited
2005
Highly Cited
2005
Background Somatic mutations in the kinase domain of the epidermal growth factor receptor tyrosine kinase gene EGFR are common in… Expand
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Highly Cited
1999
Highly Cited
1999
It has been shown previously that 4-anilino quinazolines compete with the ability of ATP to bind the epidermal growth factor… Expand
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