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BMS453

Known as: BMS 453, BMS-453 
National Institutes of Health

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Broader (1)

Retinoids

Papers overview

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2014
2014
Retinoic acid reduces migration of human breast cancer cells: role of retinoic acid receptor beta
Breast cancer is the most common malignancy in women and the appearance of distant metastases produces the death in 98% of cases… 
Highly Cited
2003
Highly Cited
2003
Retinoic acid-induced developmental defects are mediated by RARβ/RXR heterodimers in the pharyngeal endoderm
Fusion and hypoplasia of the first two branchial arches, a defect typically observed in retinoic acid (RA) embryopathy, is… 
Highly Cited
2002
Highly Cited
2002
Retinoid receptor-specific agonists alleviate experimental glomerulonephritis.
Retinoids are potent antiproliferative and anti-inflammatory compounds. We previously demonstrated that the natural pan-agonists… 
2001
2001
The retinoic acid receptor antagonist, BMS453, inhibits normal breast cell growth by inducing active TGFβ and causing cell cycle arrest
We have previously shown that a retinoic acid receptor (RAR) antagonist BMS453, which does not activate RAR-dependent gene… 
2001
2001
The retinoic acid receptor antagonist, BMS453, inhibits normal breast cell growth by inducing active TGFbeta and causing cell cycle arrest.
We have previously shown that a retinoic acid receptor (RAR) antagonist BMS453, which does not activate RAR-dependent gene… 
2001
2001
[Effects of selective RAR or/and RXR retinoids on the proliferation and differentiation of NB4 cells and their mechanisms].
OBJECTIVE To investigate the effects of RARbeta selective agonist and RARalpha antagonist (RARbeta +/RARalpha -) BMS453 in… 
1999
1999
Retinoic acid receptor antagonist BMS453 inhibits the growth of normal and malignant breast cells without activating RAR–dependent gene expression
To elucidate the role of RAR–dependent gene transcription in inhibiting breast cell growth, we have investigated the ability of… 
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