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BE 2254

Known as: 1(2H)-naphthalenone, 3,4-dihydro-2-(((2-(4-hydroxyphenyl)ethyl)amino)methyl)-, BE-2254 
National Institutes of Health

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3 relations

Broader (2)

Adrenergic alpha-AntagonistsTetralones
IBE 2254

Papers overview

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1988
1988
NO EVIDENCE OF VASCULAR DOPAMINE RECEPTORS IN THE RAT PORTAL VEIN
Summary— Dopamine (5.3 times 10−6 to 5.3 times 10−5 M) decreased the force of spontaneous myogenic contractions of the rat portal… 
1984
1984
Possible noradrenergic involvement in naloxone potentiation of apomorphine-induced stereotypic climbing in mice
1984
1984
Pipradrol-induced hyperthermia in the rabbit.
1983
1983
Characterization of postsynaptic alpha-adrenoceptors in rat aortic strips and portal veins.
Postsynaptic alpha-adrenoceptors in rat isolated aortic strips and portal veins have been examined using a number of agonist and… 
1980
1980
Amphetamine-induced increases in dopaminergic single cell firing rate after haloperidol pretreatment. Correlation with extrapyramidal side effects
1978
1978
Prevention of Estrogen-Induced Uterine Hyperemia by α-Adrenergic Receptor-Blocking Agents
Pharmacological agents were used to determine whether catecholamine receptor activation is a necessary step in the elicitation of… 
1978
1978
Interaction of 2-[beta-(4-hydroxyphenyl)ethylaminomethyl]tertralone (BE-2254: 'HEAT') with catecholamine receptors in rat brain membranes.
1976
1976
Spinal Adrenergic Mechanisms Regulating Sympathetic Outflow to Blood Vessels
The role of descending spinal catecholamine (CA)-containing fibers in cardiovascular regulation was examined. Monoamine… 
1975
1975
Central catecholamine receptor blocking actions of BE-2254 ('heat'): comparison with chlorpromazine and haloperidol.
1975
1975
Increased concentrations of 3‐methoxy‐4‐hydroxyphenylethyleneglycol and homovanillic acid in rat brain after treatment with BE‐2254 (“HEAT”)
An accelerated turnover of catecholamines in the brain produced by neuroleptics and certain other drugs has been attributed to a… 
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