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AVE 9488

Known as: AVE-9488, AVE9488 
National Institutes of Health

Papers overview

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Review
2020
Review
2020
Interplay of the red blood cell and vascular endothelial nitric oxide synthase system to combat cardiac complications of anemia
The mechanism of cardioprotection described in the clinically important study by Wischmann et al. is based on the assumption of a… 
Review
2015
Review
2015
Nitric oxide, interorganelle communication, and energy flow: a novel route to slow aging
The mitochondrial lifecycle (mitochondrial biogenesis, dynamics, and removal by mitophagy) is carefully orchestrated to ensure… 
Highly Cited
2008
Highly Cited
2008
Improvement in Left Ventricular Remodeling by the Endothelial Nitric Oxide Synthase Enhancer AVE9488 After Experimental Myocardial Infarction
Background— Reduced endothelial nitric oxide (NO) bioavailability contributes to the progression of heart failure. In this study… 
2008
2008
Enhancer AVE9488 After Experimental Myocardial Infarction Improvement in Left Ventricular Remodeling by the Endothelial Nitric Oxide Synthase
2008
2008
Abstract 5390: The Endothelial Nitric Oxide Synthase Enhancer AVE9488 Increases Circulating Endothelial Progenitor Cells and Prevents Heart Failure Early after Myocardial Infarction
We studied the effects of AVE9488, a novel endothelial nitric oxide synthase (eNOS) expression enhancer on endothelial progenitor… 
2008
2008
Treatment of bile duct‐ligated rats with the nitric oxide synthase transcription enhancer AVE 9488 ameliorates portal hypertension
Aim: Nitric oxide levels are decreased in the cirrhotic liver and increased in the systemic vasculature. We investigated whether… 
2006
2006
Abstract 486: Targeting Bone Marrow Endothelial Nitric Oxide Synthase (eNOS) by the eNOS Enhancer AVE9488 Increases Circulating Endothelial Progenitor Cells After Myocardial Infarction
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