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2,3-dimethoxy-1,4-naphthoquinone

Known as: DMNQ 
National Institutes of Health

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Broader (1)

Naphthoquinones

Papers overview

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Highly Cited
2011
Highly Cited
2011
Differentiation of SH-SY5Y cells to a neuronal phenotype changes cellular bioenergetics and the response to oxidative stress.
Highly Cited
2010
Highly Cited
2010
Mitochondrial reserve capacity in endothelial cells: The impact of nitric oxide and reactive oxygen species.
Highly Cited
2009
Highly Cited
2009
Resveratrol reduces endothelial oxidative stress by modulating the gene expression of superoxide dismutase 1 (SOD1), glutathione peroxidase 1 (GPx1) and NADPH oxidase subunit (Nox4).
Resveratrol, an important antioxidant found in grapes and wine, is likely to contribute to red wine's potential to prevent human… 
Highly Cited
2004
Highly Cited
2004
Variable regulation of glutamate cysteine ligase subunit proteins affects glutathione biosynthesis in response to oxidative stress.
Review
2003
Review
2003
Neural stem cells and cell death.
Highly Cited
1997
Highly Cited
1997
Nitric oxide and superoxide induced p53 and Bax accumulation during mesangial cell apoptosis.
During proliferative glomerulonephritis, the early phase of mesangiolysis is linked to increased nitric oxide (NO) production. NO… 
Highly Cited
1995
Highly Cited
1995
Interleukin 6 production by lipopolysaccharide-stimulated human fibroblasts is potently inhibited by naphthoquinone (vitamin K) compounds.
Naphthoquinone vitamins (vitamins K) are widely recognized for their role in the gamma-carboxylation of specific glutamyl… 
Highly Cited
1994
Highly Cited
1994
Quinone-induced oxidative stress elevates glutathione and induces gamma-glutamylcysteine synthetase activity in rat lung epithelial L2 cells.
Highly Cited
1994
Highly Cited
1994
Different prooxidant levels stimulate growth, trigger apoptosis, or produce necrosis of insulin-secreting RINm5F cells. The role of intracellular polyamines.
Highly Cited
1988
Highly Cited
1988
Redox cycling and sulphydryl arylation; their relative importance in the mechanism of quinone cytotoxicity to isolated hepatocytes.
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