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1-methylanthracene

National Institutes of Health

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1 relation

Broader (1)

Anthracenes

Papers overview

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2019
2019
Dysregulation of Gap Junction Function and Cytokine Production in Response to Non-Genotoxic Polycyclic Aromatic Hydrocarbons in an In Vitro Lung Cell Model
Polycyclic aromatic hydrocarbons (PAHs), prevalent contaminants in our environment, in many occupations, and in first and second… 
2019
2019
Early Mechanistic Events Induced by Low Molecular Weight Polycyclic Aromatic Hydrocarbons in Mouse Lung Epithelial Cells: A Role for Eicosanoid Signaling.
Low molecular weight polycyclic aromatic hydrocarbons (LMW PAHs; < 206.3 g/mol) are under regulated environmental contaminants… 
2014
2014
Theoretical study of the molecular properties of dimethylanthracenes as properties for the prediction of their biodegradation and mutagenicity.
2013
2013
Polycyclic Aromatic Hydrocarbon-Induced Signaling Events Relevant to Inflammation and Tumorigenesis in Lung Cells Are Dependent on Molecular Structure
Polycyclic aromatic hydrocarbons (PAHs) are ubiquitous environmental and occupational toxicants, which are a major human health… 
2007
2007
Cigarette smoke components inhibited intercellular communication and differentiation in human pancreatic ductal epithelial cells
Smoking is a well‐documented risk factor for the development of pancreatic adenocarcinoma. Although the most abundant polycyclic… 
2007
2007
Comprehensive photoelectron spectroscopic study of anionic clusters of anthracene and its alkyl derivatives: electronic structures bridging molecules to bulk.
The evolution of the electronic structure of molecular aggregates is investigated using anion photoelectron (PE) spectroscopy for… 
2005
2005
Internal rotation of methyl group in 2- and 1-methylanthracene studied by electronic spectroscopy and DFT calculations
Highly Cited
2002
Highly Cited
2002
Polycyclic aromatic hydrocarbons present in cigarette smoke cause endothelial cell apoptosis by a phospholipase A2dependent mechanism
Smoking is a major risk factor for endothelial cell injury and subsequent coronary artery disease. Epidemiological studies… 
Highly Cited
1999
Highly Cited
1999
Polycyclic aromatic hydrocarbons with bay-like regions inhibited gap junctional intercellular communication and stimulated MAPK activity.
Many polycyclic aromatic hydrocarbons (PAHs) are known carcinogens. A considerable amount of research has been devoted to… 
Highly Cited
1998
Highly Cited
1998
Bay or baylike regions of polycyclic aromatic hydrocarbons were potent inhibitors of Gap junctional intercellular communication.
Many polycyclic aromatic hydrocarbons (PAHs) are known carcinogens, and a considerable amount of research has been devoted to… 
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