1-methylanthracene

 
National Institutes of Health

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Anthracenes

Papers overview

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2017
2017
Secondhand Smoke-Prevalent Polycyclic Aromatic Hydrocarbon Binary Mixture-Induced Specific Mitogenic and Pro-inflammatory Cell Signaling Events in Lung Epithelial Cells.
Low molecular weight polycyclic aromatic hydrocarbons (LMW PAHs; < 206.3 g/mol) are prevalent and ubiquitous environmental… (More)
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2017
2017
Environmentally prevalent polycyclic aromatic hydrocarbons can elicit co-carcinogenic properties in an in vitro murine lung epithelial cell model
Low molecular weight (LMW) polycyclic aromatic hydrocarbons (PAH) are the most abundant PAHs environmentally, occupationally, and… (More)
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2013
2013
Polycyclic Aromatic Hydrocarbon-Induced Signaling Events Relevant to Inflammation and Tumorigenesis in Lung Cells Are Dependent on Molecular Structure
Polycyclic aromatic hydrocarbons (PAHs) are ubiquitous environmental and occupational toxicants, which are a major human health… (More)
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2008
2008
Tumor promoting properties of a cigarette smoke prevalent polycyclic aromatic hydrocarbon as indicated by the inhibition of gap junctional intercellular communication via phosphatidylcholine-specific…
Inhibition of gap junctional intercellular communication (GJIC) and the activation of intracellular mitogenic pathways are common… (More)
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2007
2007
Cigarette smoke components inhibited intercellular communication and differentiation in human pancreatic ductal epithelial cells.
Smoking is a well-documented risk factor for the development of pancreatic adenocarcinoma. Although the most abundant polycyclic… (More)
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2007
2007
Comprehensive photoelectron spectroscopic study of anionic clusters of anthracene and its alkyl derivatives: electronic structures bridging molecules to bulk.
The evolution of the electronic structure of molecular aggregates is investigated using anion photoelectron (PE) spectroscopy for… (More)
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2005
2005
Internal rotation of methyl group in 2- and 1-methylanthracene studied by electronic spectroscopy and DFT calculations
The S1–S0 fluorescence excitation and dispersed fluorescence spectra of 2and 1-methylanthracene are measured in a supersonic free… (More)
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2002
2002
Polycyclic aromatic hydrocarbons present in cigarette smoke cause endothelial cell apoptosis by a phospholipase A2-dependent mechanism.
Smoking is a major risk factor for endothelial cell injury and subsequent coronary artery disease. Epidemiological studies… (More)
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1999
1999
Polycyclic aromatic hydrocarbons with bay-like regions inhibited gap junctional intercellular communication and stimulated MAPK activity.
Many polycyclic aromatic hydrocarbons (PAHs) are known carcinogens. A considerable amount of research has been devoted to… (More)
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1998
1998
Bay or baylike regions of polycyclic aromatic hydrocarbons were potent inhibitors of Gap junctional intercellular communication.
Many polycyclic aromatic hydrocarbons (PAHs) are known carcinogens, and a considerable amount of research has been devoted to… (More)
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