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polyglutamine

Known as: poly(glutamine), polyQ 
National Institutes of Health

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1 relation

Broader (1)

Peptides

Papers overview

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Highly Cited
2010
Highly Cited
2010
Human ataxias: a genetic dissection of inositol triphosphate receptor (ITPR1)-dependent signaling
Highly Cited
2007
Highly Cited
2007
"Prion-proof" for [PIN+]: infection with in vitro-made amyloid aggregates of Rnq1p-(132-405) induces [PIN+].
Highly Cited
2007
Highly Cited
2007
Reconsidering the mechanism of polyglutamine peptide aggregation.
There are at least nine neurodegenerative diseases associated with proteins that contain an unusually expanded polyglutamine… 
Highly Cited
2007
Highly Cited
2007
Properties of human Cav2.1 channel with a spinocerebellar ataxia type 6 mutation expressed in Purkinje cells
Review
2006
Review
2006
Pathogenesis, animal models and therapeutics in Spinal and bulbar muscular atrophy (SBMA)
Highly Cited
2005
Highly Cited
2005
Modulation of age at onset of Huntington disease patients by variations in TP53 and human caspase activated DNase (hCAD) genes
Highly Cited
2005
Highly Cited
2005
Characterization of a possible amyloidogenic precursor in glutamine-repeat neurodegenerative diseases.
Several neurodegenerative diseases are linked to expanded repeats of glutamine residues, which lead to the formation of amyloid… 
Highly Cited
2004
Highly Cited
2004
Effect of tissue transglutaminase on the solubility of proteins containing expanded polyglutamine repeats
The expansion of a polyglutamine (polyQ) domain in neuronal proteins is the molecular genetic cause of at least eight… 
Review
1998
Review
1998
Genes involved in hereditary ataxias
Highly Cited
1998
Highly Cited
1998
Evolution of Simple Repeats in DNA and Their Relation to Human Disease
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